Amelogenin is a Cell Adhesion Protein
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概要
- 論文の詳細を見る
- 2002-07-01
著者
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SIMMER J.
Department of Biologic and Materials Science, University of Michigan School of Dentistry, Dental Res
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Ryu O.
Department Of Pediatric Dentistry University Of Texas Health Science Center
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Simmer J.
Department Of Biologic And Materials Science University Of Michigan School Of Dentistry Dental Resea
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Simmer J.
Department Of Pediatric Dentistry University Of Texas Health Science Center
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COCHRAN D.
Department of Periodontics, University of Texas Health Science Center at San Antonio
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HOANG A.
Department of Periodontics, University of Texas Health Science Center
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KLEBE R.
Department of Cellular and Structural Biology, University of Texas Health Science Center
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STEFFENSEN B.
Department of Periodontics, University of Texas Health Science Center
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Cochran D.
Department Of Periodontics University Of Texas Health Science Center
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Klebe R.
Department Of Cellular And Structural Biology University Of Texas Health Science Center
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Steffensen B.
Department Of Periodontics University Of Texas Health Science Center
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Hoang A.
Department Of Periodontics University Of Texas Health Science Center
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COCHRAN D.
Department of Periodontics
関連論文
- Premature Stop Codon in MMP20 Causing Amelogenesis Imperfecta
- Processing of Ameloblastin by MMP-20
- Hereditary Dentin Defects
- Fam83h is Associated with Intracellular Vesicles and ADHCAI
- Enhanced Bone Apposition to a Chemically Modified SLA Titanium Surface
- Amelogenin is a Cell Adhesion Protein
- Amelogenin p. M1T and p. W4S Mutations Underlying Hypoplastic X-linked Amelogenesis Imperfecta
- Mmp-20 and Klk4 Cleavage Site Preferences for Amelogenin Sequences
- DPPI May Activate KLK4 during Enamel Formation
- Cleavage Site Specificity of MMP-20 for Secretory-stage Ameloblastin
- Splicing Determines the Glycosylation State of Ameloblastin
- Peri-implant Inflammation Defined by the Implant-Abutment Interface
- Overlapping DSPP Mutations Cause Dentin Dysplasia and Dentinogenesis Imperfecta
- Altered Enamelin Phosphorylation Site Causes Amelogenesis Imperfecta
- ENAM Mutations in Autosomal-dominant Amelogenesis Imperfecta
- Novel WDR72 Mutation and Cytoplasmic Localization
- Regulation of Dental Enamel Shape and Hardness
- Transgenic Rescue of Enamel Phenotype in Ambn Null Mice
- Novel MSX1 Frameshift Causes Autosomal-dominant Oligodontia
- PDGF-α Receptor Subunit Expression Down-regulated by IL-1β in Human Periodontal Ligament Cells
- FAM83H Mutations Cause ADHCAI and Alter Intracellular Protein Localization
- Inflammation and Uncoupling as Mechanisms of Periodontal Bone Loss