Loss of the pro-apoptotic BH3-only Bcl-2 family member Bim sustains B lymphopoiesis in the absence of IL-7
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概要
- 論文の詳細を見る
- 2009-06-01
著者
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Strasser Andreas
Walter And Eliza Hall Inst. Of Medical Res. Victoria Aus
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Strasser Andreas
Molecular Genetics Of Cancer Division The Walter And Eliza Hall Institute Of Medical Research
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HUNTINGTON Nicholas
Cytokines and Lymphoid Development Unit, Institut Pasteur
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LABI Verena
Division of Developmental Immunology, Department of Biocenter, Innsbruck Medical University
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CUMANO Ana
Inserm U668
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VIEIRA Paulo
Inserm U668
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VILLUNGER Andreas
Division of Developmental Immunology, Department of Biocenter, Innsbruck Medical University
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DI SANTO
Cytokines and Lymphoid Development Unit, Institut Pasteur
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ALVES Nuno
Cytokines and Lymphoid Development Unit, Institut Pasteur
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Di Santo
Cytokines And Lymphoid Development Unit Institut Pasteur
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Alves Nuno
Cytokines And Lymphoid Development Unit Institut Pasteur
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Labi Verena
Division Of Developmental Immunology Department Of Biocenter Innsbruck Medical University
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Huntington Nicholas
Cytokines And Lymphoid Development Unit Institut Pasteur
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Villunger Andreas
Division Of Developmental Immunology Department Of Biocenter Innsbruck Medical University
関連論文
- Loss of the pro-apoptotic BH3-only Bcl-2 family member Bim sustains B lymphopoiesis in the absence of IL-7
- Autoimmune kidney disease and lymphadenopathy in NODIpr mice are not modified by deficiency in tumor necrosis factor receptor 1 or β_2-microglobulin
- Overexpression of Bcl-2 does not rescue impaired B lymphopoiesis in IL-7 receptor-deficient mice but can enhance survival of mature B cells