Autoimmune kidney disease and lymphadenopathy in NODIpr mice are not modified by deficiency in tumor necrosis factor receptor 1 or β_2-microglobulin
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概要
- 論文の詳細を見る
- 2003-06-01
著者
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Strasser Andreas
Walter And Eliza Hall Inst. Of Medical Res. Victoria Aus
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Strasser Andreas
Walter And Eliza Hall Institute Of Medical Research Po Royal Melbourne Hospital
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Stockwell Dina
Department Of Microbiology And Immunology University Of Melbourne
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Allison J
Royal Melbourne Hospital Victoria Aus
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CATTERALL Tara
Department of Microbiology and Immunology, University of Melbourne
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MARSHALL Vikki
Walter and Eliza Hall Institute of Medical Research, PO Royal Melbourne Hospital
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ALLISON Janette
Department of Microbiology and Immunology, University of Melbourne
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Marshall Vikki
Walter And Eliza Hall Institute Of Medical Research Po Royal Melbourne Hospital
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Catterall Tara
Department Of Microbiology And Immunology University Of Melbourne
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Allison Janette
Department Of Microbiology And Immunology University Of Melbourne
関連論文
- Loss of the pro-apoptotic BH3-only Bcl-2 family member Bim sustains B lymphopoiesis in the absence of IL-7
- Autoimmune kidney disease and lymphadenopathy in NODIpr mice are not modified by deficiency in tumor necrosis factor receptor 1 or β_2-microglobulin
- Overexpression of Bcl-2 does not rescue impaired B lymphopoiesis in IL-7 receptor-deficient mice but can enhance survival of mature B cells
- PERFORIN- AND FAS-MEDIATED KILLING BY CTL ARE BOTH BLOCKED BY SUPPRESSOR OF CYTOKINE SIGNALLING-1 OVEREXPRESSION IN PANCREATIC BETA CELLS