Absence of Smad3 confers radioprotection through modulation of ERK-MAPK in primary dermal fibroblasts
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概要
- 論文の詳細を見る
- 2007-10-01
著者
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Cook John
Radiation Biology Branch, National Cancer Institute, National Institutes of Health
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Mitchell James
Radiation Biology Branch, National Cancer Institute, National Institutes of Health
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DEGRAFF William
Radiation Biology Branch, Center for Cancer Research, National Cancer Institute, NIH
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Cook John
Radiation Biology Branch National Cancer Institute National Institutes Of Health
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Cook John
Radiation Biology National Cancer Institute National Institutes Of Health
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Mitchell James
Radiation Biology National Cancer Institute National Institutes Of Health
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Mitchell James
Radiation Biology Branch Center For Cancer Research National Cancer Institute Nih
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Degraff William
Radiation Biology National Cancer Institute National Institutes Of Health
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Degraff William
Radiation Biology Branch Center For Cancer Research National Cancer Institute Nih
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ARANY Praveen
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, National Institutes of
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FLANDERS Kathleen
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, National Institutes of
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ROBERTS Anita
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, National Institutes of
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Roberts Anita
Laboratory Of Cell Regulation And Carcinogenesis National Cancer Institute
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Arany Praveen
Laboratory Of Cell Regulation And Carcinogenesis National Cancer Institute National Institutes Of He
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Roberts Anita
Laboratory Of Cell Regulation And Carcinogenesis National Cancer Institute National Institutes Of He
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Flanders Kathleen
Laboratory Of Cell Regulation And Carcinogenesis National Cancer Institute National Institutes Of He
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Flanders Kathleen
Laboratory Of Cell Regulation And Carcinogenesis National Cancer Institute
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- Absence of Smad3 confers radioprotection through modulation of ERK-MAPK in primary dermal fibroblasts
- A diffusing sphere which delivers homogeneous laser light for use in photodynamic therapy
- Role of Smad3 in the hormonal modulation of in vivo wound healing responses
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