PAR-2 Deficient CD4^+ T Cells Exhibit Downregulation of IL-4 and Upregulation of IFN-γ after Antigen Challenge in Mice
スポンサーリンク
概要
- 論文の詳細を見る
- 2006-09-01
著者
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Bacon Kevin
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Bacon Kevin
バイエル薬品
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SHICHIJO Michitaka
Respiratory Diseases Research, Research Center Kyoto, Bayer Yakuhin, Ltd.
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KONDO Shinichi
Respiratory Diseases Research, Research Center Kyoto, Bayer Yakuhin, Ltd.
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ISHIMORI Mina
Respiratory Diseases Research, Research Center Kyoto, Bayer Yakuhin, Ltd.
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WATANABE Shinichi
Respiratory Diseases Research, Research Center Kyoto, Bayer Yakuhin, Ltd.
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HELIN Heidi
Respiratory Diseases Research, Research Center Kyoto, Bayer Yakuhin, Ltd.
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YAMASAKI Tsugiko
Respiratory Diseases Research, Research Center Kyoto, Bayer Yakuhin, Ltd.
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STEVENS Mary
Lawrence Berkeley National Lab, University of California
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GANTNER Florian
Respiratory Diseases Research, Research Center Kyoto, Bayer Yakuhin, Ltd.
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Helin Heidi
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Stevens Mary
Lawrence Berkeley National Lab University Of California
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Ishimori Mina
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Gantner Florian
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Yamasaki Tsugiko
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Shichijo Michitaka
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Kondo Shinichi
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Watanabe Shinichi
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
関連論文
- 好塩基球のPGD2受容体発現とその機能
- CRTH2 is a prominent effector in contact hypersensitivity-induced neutrophil inflammation
- Essential role of MHC II-independent CD4^+ T cells, IL-4 and STAT6 in contact hypersensitivity induced by fluorescein isothiocyanate in the mouse
- ラマトロバンのCRTH2の拮抗作用
- PAR-2 Deficient CD4^+ T Cells Exhibit Downregulation of IL-4 and Upregulation of IFN-γ after Antigen Challenge in Mice