CRTH2 is a prominent effector in contact hypersensitivity-induced neutrophil inflammation
スポンサーリンク
概要
- 論文の詳細を見る
- 2004-07-01
著者
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Bacon Kevin
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Bacon Kevin
バイエル薬品
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Gantner Florian
Bayer Yakuhin Ltd. Research Center Kyoto Department Of Respiratory Diseases Research
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TAKESHITA Keisuke
Bayer Yakuhin, Ltd., Research Center Kyoto, Respiratory Disease Research
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YAMASAKI Tsugiko
Bayer Yakuhin, Ltd., Research Center Kyoto, Respiratory Disease Research
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NAGAO Koichi
Bayer Yakuhin, Ltd., Research Center Kyoto, Respiratory Disease Research
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SUGIMOTO Hiromi
Bayer Yakuhin, Ltd., Research Center Kyoto, Respiratory Disease Research
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SHICHIJO Michitaka
Bayer Yakuhin, Ltd., Research Center Kyoto, Respiratory Disease Research
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BACON Kevin
Bayer Yakuhin, Ltd., Research Center Kyoto, Respiratory Disease Research
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Bacon Kevin
Bayer Yakuhin Ltd. Research Center Kyoto Department Of Respiratory Diseases Research
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SHICHIJO Michitaka
Respiratory Diseases Research, Research Center Kyoto, Bayer Yakuhin, Ltd.
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Nagao Koichi
Bayer Yakuhin Ltd. Research Center Kyoto Respiratory Disease Research
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Sugimoto Hiromi
Bayer Yakuhin Ltd. Research Center Kyoto Respiratory Disease Research
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Yamasaki Tsugiko
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
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Takeshita Keisuke
Bayer Yakuhin Ltd. Research Center Kyoto Department Of Respiratory Diseases Research
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Shichijo Michitaka
Respiratory Diseases Research Research Center Kyoto Bayer Yakuhin Ltd.
関連論文
- 好塩基球のPGD2受容体発現とその機能
- CRTH2 is a prominent effector in contact hypersensitivity-induced neutrophil inflammation
- Essential role of MHC II-independent CD4^+ T cells, IL-4 and STAT6 in contact hypersensitivity induced by fluorescein isothiocyanate in the mouse
- ラマトロバンのCRTH2の拮抗作用
- PAR-2 Deficient CD4^+ T Cells Exhibit Downregulation of IL-4 and Upregulation of IFN-γ after Antigen Challenge in Mice