Pancreatic Dysfunction in Cystic Fibrosis Occurs as a Result of Impairments in Luminal pH, Apical Trafficking of Zymogen Granule Membranes, and Solubilization of Secretory Enzymes
スポンサーリンク
概要
- 論文の詳細を見る
- 1996-01-01
著者
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Kern Horst
Department Of Cell Biology And Cytopathology Philipps University
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Scheele George
Laboratory Of Cell And Molecular Biology Charles A. Dana Research Institute Thorndike Laboratory
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FUKUOKA Shin-Ichi
Research Institute for Food Science, Kyoto University
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FREEDMAN Steven
Laboratory of Cell and Molecular Biology, Charles A. Dana Research Institute, Thorndike Laboratory
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Fukuoka Shin-ichi
Research Institute For Food Science Kyoto University
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Freedman Steven
Laboratory Of Cell And Molecular Biology Charles A. Dana Research Institute Thorndike Laboratory
関連論文
- Expression of Bcl-2 and PCNA in Duct Cells After Pancreatic Duct Ligation in Rats
- Molecular basis for pancreatic dysfunction in cystic fibrosis
- Pancreatic Dysfunction in Cystic Fibrosis Occurs as a Result of Impairments in Luminal pH, Apical Trafficking of Zymogen Granule Membranes, and Solubilization of Secretory Enzymes
- Increase in Lysophosphatidylethanolamine in the Cell Membrane upon the Regulated Exocytosis of Pancreatic Acinar AR42J Cells
- High-Level Secretion of Native Rat Pancreatic Lithostathine in a Baculovirus Expression System
- Analysis of ZAPs, Zymogen Granule Membrane Associated Proteins, in the Regulated Exocytosis of the Pancreas