Wound epithelialization deficits in the transforming growth factor-α knockout mouse
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概要
- 論文の詳細を見る
- 2001-09-01
著者
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Mustoe Thomas
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Northwestern Univ
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Mustoe Thomas
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Feinberg School O
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Chao Jerome
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Northwestern Univ
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Mogford Jon
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Northwestern Univ
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Mogford Jon
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Feinberg School O
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KIM Injoong
Wound Healing Research Laboratory, Division of Plastic & Reconstructive Surgery, Northwestern Univer
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Kim Injoong
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Northwestern Univ
関連論文
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- Use of hypoxia-inducible factor signal transduction pathway to measure O_2 levels and modulate growth factor pathways
- Inhibition of prolyl 4-hydroxylase reduces scar hypertrophy in a rabbit model of cutaneous scarring
- Wound epithelialization deficits in the transforming growth factor-α knockout mouse
- Quantifying tissue level ischemia : Hypoxia response element-luciferase transfection in a rabbit ear model
- Delayed wound healing in Mac-1-deficient mice is associated with normal monocyte recruitment