Mogford Jon | Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Feinberg School O
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概要
- MOGFORD JON E.の詳細を見る
- 同名の論文著者
- Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Feinberg School Oの論文著者
関連著者
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Mustoe Thomas
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Feinberg School O
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Mogford Jon
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Feinberg School O
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Chao Jerome
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Northwestern Univ
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Kim Injoong
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Northwestern Univ
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Mustoe Thomas
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Northwestern Univ
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Mogford Jon
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Northwestern Univ
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ROY NAKSHATRA
Wound Healing Research Laboratory, Division of Plastic & Reconstructive Surgery, Feinberg School of
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CROSS KEVIN
Wound Healing Research Laboratory, Division of Plastic & Reconstructive Surgery, Feinberg School of
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KIM Injoong
Wound Healing Research Laboratory, Division of Plastic & Reconstructive Surgery, Northwestern Univer
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Cross Kevin
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Feinberg School O
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Roy Nakshatra
Wound Healing Research Laboratory Division Of Plastic & Reconstructive Surgery Feinberg School O
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MUSTOE Thomas
Wound Healing Research Laboratory, Division of Plastic Surgery, Northwestern University Feinberg School of Medicine
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SISCO Mark
Wound Healing Research Laboratory, Division of Plastic Surgery, Northwestern University Feinberg School of Medicine
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MAYADAS Tanya
Department of Pathology, Center for Excellence in Vascular Biology, Brigham and Women's Hospital and Harvard Medical School
著作論文
- Use of hypoxia-inducible factor signal transduction pathway to measure O_2 levels and modulate growth factor pathways
- Wound epithelialization deficits in the transforming growth factor-α knockout mouse
- Delayed wound healing in Mac-1-deficient mice is associated with normal monocyte recruitment