NKT lymphocyte ontogeny and function are impaired in low antibody-producer Biozzi mice: gene mapping in the interval-specific congenic strains raised for immunomodulatory genes
スポンサーリンク
概要
著者
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KOEZUKA Yasuhiko
Pharmaceutical Research Laboratory, Kirin Brewery Co., Ltd.
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Herbelin A
Inserm U25 Centre Claude Bernard Hopital Necker
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Bach Jean-francois
Inserm U25 Centre Claude Bernard Hopital Necker
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ARAUJO Luiza
INSERM U255, Institut Curie
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PUEL Anne
INSERM U255, Institut Curie
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GOUARIN Christine
INSERM U25, Centre Claude Bernard, Hopital Necker
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HAMEG Agathe
INSERM U25, Centre Claude Bernard, Hopital Necker
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MEVEL Jean-Claude
INSERM U255, Institut Curie
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MOUTON Denise
INSERM U255, Institut Curie
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HERBELIN Andre
INSERM U25, Centre Claude Bernard, Hopital Necker
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Puel Anne
Inserm U255 Institut Curie
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Araujo Luiza
Inserm U255 Institut Curie
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Hameg Agathe
Inserm U25 Centre Claude Bernard Hopital Necker
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Mouton Denise
Inserm U255 Institut Curie
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Herbelin Andre
Inserm U25 Centre Claude Bernard Hopital Necker
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Gouarin Christine
Inserm U25 Centre Claude Bernard Hopital Necker
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Mevel Jean-claude
Inserm U255 Institut Curie
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Koezuka Yasuhiko
Pharmaceutical Research Laboratory Kirin Brewery Co
関連論文
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- Molecular analysis of TCR junctional variability in individual infiltrated islets of non-obese diabetic mice: evidence for the constitution of largely autonomous T cell foci within the same pancreas
- NKT lymphocyte ontogeny and function are impaired in low antibody-producer Biozzi mice: gene mapping in the interval-specific congenic strains raised for immunomodulatory genes
- Requirement of IL-7 for IL-4-producing potential of MHC class I-selected CD4^-CD8^- TCRαβ^+ thymocytes
- IL-7 reverses NK1^+ T cell-defective IL-4 production in the non-obese diabetic mouse
- NK1.1^+ T cells from IL-7-deficient mice have a normal distribution and selection but exhibit impaired cytokine production