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Laboratory Of Molecular Pharmacology Biosignal Research Center Kobe University | 論文
- Inflammatory Cytokine Tumor Necrosis Factor-α Enhances Nerve Growth Factor Production in Human Keratinocytes, HaCaT Cells
- A Single Stranded DNA-Binding Protein, SSCRE-BP/Pura, in Rat Lung and Its Increase in Allergic Airway Inflammation
- Congo Red, an Amyloid-Inhibiting Compound, Alleviates Various Types of Cellular Dysfunction Triggered by Mutant Protein Kinase Cγ That Causes Spinocerebellar Ataxia Type 14 (SCA14) by Inhibiting Oligomerization and Aggregation
- Electrophysiological and Pharmacological Characteristics of lonotropic Glutamate Receptors in Medial Vestibular Nucleus Neurons: A Whole Cell Patch Clamp Study in Acutely Dissociated Neurons
- Spatiotemporal Analysis of the Molecular Interaction between PICK1 and PKC
- P2-08 Visualization of intracellular signaling system : immunogold electron microscopic study on γPKC translocation
- A-1 Immunogold electron microscopic studies of subtype- and stimulus-specific PKC translocation
- W2-1 Analysis of PKC targeting mechanism in living cells for understanding PKC signaling
- Visualization of stimulus-and subtype-specific translocation of protein kinase C(PKC)in living cells : Molecular mechanism of PKC targeting
- Subspecies-Specific Targeting Mechanism of Protein Kinase C
- Immunohistochemical localization of serotonin transporter in normal and colchicine treated rat brain
- Effects of tricyclic and tetracyclic antidepressants on the three subtypes of GABA transporter
- Congo Red, an Amyloid-Inhibiting Compound, Alleviates Various Types of Cellular Dysfunction Triggered by Mutant Protein Kinase Cγ That Causes Spinocerebellar Ataxia Type 14 (SCA14) by Inhibiting Oligomerization and Aggregation
- Visualisation of intracellular signaling using GFP-fusion protein
- Elucidation of the Molecular Mechanism and Exploration of Novel Therapeutics for Spinocerebellar Ataxia Caused by Mutant Protein Kinase Cγ