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First Department Of Internal Medicine Fukushima Medical University | 論文
- Venous Thromboembolism : Deep Vein Thrombosis With Pulmonary Embolism, Deep Vein Thrombosis Alone, and Pulmonary Embolism Alone(Hypertension/Pulmonary Circulation)
- PJ-743 Incidence and Clinical Characteristics of Venous Thromboembolism(Pulmonary circulation(04)(H),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-114 Incidence of Pulmonary Embolism in Psychiatric Wards in Japan(Thromboembolism/Antithrombotic therapy/Thrombolysis(01)(H),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- Pulmonary embolism is an important cause of death in young adults
- Inhibition of Matrix Metalloproteases (MMPs) Suppresses Rac/NADPH Oxidase-derived Reactive Oxygen Species (ROS) Generation in Oxidized Low Density Lipoprotein-stimuleted Endothelial Cells(Atherosclerosis, Basic 4 (IHD), The 69th Annual Scientific Meeting
- Therapeutic Effect of Intravenous Administration of Statin on eNOS Downregulation and ROS Generation in Response to Thrombin in Vivo(Cardiovascular Pharmacology, Basic/Clinical 2 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-582 Intravenous administration of statin prevents RhoA activation-triggered eNOS downregulation and reactive oxygen species induced by thrombin in rats(Cardiovascular Pharmacology, Basic/Clinical 5 (H) : PJ98)(Poster Session (Japanese))
- PE-216 Mechanism and Role of Geranylgeranylation in RhoA Activation in Endothelial Dysfunction(Metabolism/Biochemistry/Energetics 1 (IHD) : PE37)(Poster Session (English))
- OE-118 Inhibition of Matrix Metalloproteinases (MMPs) Suppresses the RhoA-dependent eNOS Downergulation Induced by Oxidized LDL in Endothelial Cells(Atherosclerosis, Basic 1 (IHD) : OE14)(Oral Presentation (English))
- Rho Activation and Extracellular Signal-Regulated Kinase (ERK) Downregulation Are Crucial for eNOS Downregulation in Monocyte Adhesion to Endothelial Cells
- Thrombin-lnduced Downregulation of Endothelial Nitric Oxide Synthase (eNOS) via Rho Activation in Human Endothelial Cells :Involvement of Geranylgeranyl Transferase Activation
- Thrombin-Stimulated Ca^ Signaling Mediated via RhoA Activationin Tissue Factor (TF) and Plasminogen Activator Inhibitor-1 (PAI-1) Synthesis in Endothelial Cells
- RhoA Activation Plays an Integral Role in Tissue Factor (TF) Gene Expression
- Oxidized LDL Downregulates Endothelial Nitric Oxide Synthase (eNOS) via Rapid RhoA Activaion in Endothelial Cells
- OJ-193 Carbon Monoxide from Heme Oxygenase-1 Attenuates Neointimal Formation and Inflammatory Responses in Carotid Artery Ligation Model of LDL-receptor Knockout Mice(Atherosclerosis, Basic 2 (IHD) : OJ22)(Oral Presentation (Japanese))
- Heme Oxygenase-1 Deficiency Increases the Generations of Reactive Oxygen Species and the Expression of Plasminogen Activator Inhibitor-1
- Carbon Monoxide from Heme Oxygenase-1 Scavenges Reactive Oxygen Species Generated by Oxidized LDL in Macrophages through P38 MAP Kinase Pathway
- RELATIONSHIP OF THALLIUM-201 DEFECT AND LEFT VENTRICULAR FUNCTION AFTER DIPYRIDAMOLE INFUSION
- Quantitative Assessment of Myocardial ^Tc-sestamibi Uptake During Exercise : Usefulness of Response Rate for Assessing Severity of Coronary Artery Disease
- Unusual Pulmonary Embolism : Septic Pulmonary Embolism and Amniotic Fluid Embolism
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