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Department of Cardiovascular Medicine, University of Tokyo Hospital | 論文
- Connexin-43 Redistribution and Gap Junction Activation During Forced Restraint Protects Against Sudden Arrhythmic Death in Rats
- The Significant Association of Genetic Polymorphisms in the KLF5 Gene with Coronary Artery Disease(Atherosclerosis, Clinical 9 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-447 The genetic polymorphisms in the insulin-resistance related genes as coronary risks(Preventive Medicine/Epidemiology/Education 4 (H) : PJ75)(Poster Session (Japanese))
- Systemic Delivery of Retinoic Acid Receptor α Specific Agonist Inhibits In-Stent Restenosis by Inducing Differentiation of Smooth Muscle Cells(Vascular Smooth Muscle 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Kruppel-like Transcription Factor KLF5 is a Key Regulator Governing Adipocyte Differentiation(Metabolic Syndrome (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Serum Response Factor (SRF) Cofactors in Smooth Muscle Differentiation
- The Significant Association of the Genetic Polymorphisms of Werner Helicase and β2 Adrenergic Receptor with Susceptibility to Coronary Heart Disease
- OJ-512 A Zinc Finger Transcription factor δ EF1 Controls Smooth Muscle Cell Differentiation during Development and in Vascular Diseases(Vascular Smooth Muscle 2 (H) : OJ63)(Oral Presentation (Japanese))
- OE-161 Oral Retinoic Acid Receptor Alpha Agonist Inhibits In-Stent Restenosis(Restenosis after Angioplasty, Basic/Clinical 1 (IHD) : OE20)(Oral Presentation (English))
- Angiotensin II and Transforming Growth Factor Beta Induced Angiogenesis Associated Mts1 in Smooth Muscle Cells
- PPAR γ Ligand Dependent Interaction with Transcriptional Coactivators Control the Vascular Smooth Muscle Cell Differentiation
- An E-Box Binding Transcription Factor Controls Smooth Muscle Cell Differentiation Marker Genes
- Kruppel Like Zinc-Finger Trascription Factor 5 (KLF5) Madiate Immune Responses in Cardiovascular Remodeling
- Kruppel-Like Factor 5 (KLF5) and Peroxisome Proliferatesγ (PPARγ) Cooperatively Control Cellular Responses in Vascular Diseases
- Restraint Stress Induces Connexin-43 Translocation via α-Adrenoceptors in Rat Heart
- Combination Evaluation of Preoperative Risk Indices Predicts Requirement of Biventricular Assist Device