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Department Of Pharmacology Graduate School Of Pharmaceutical Sciences Kyoto University | 論文
- Involvement of TRPM7 in Cell Growth as a Spontaneously Activated Ca^ Entry Pathway in Human Retinoblastoma Cells
- Current Perspective : Dissecting Receptor-Mediated Ca^ Influx Pathways : TRP Channels and Their Native Counterparts
- Receptor-Mediated Modulation of Voltage-Dependent Ca^ Channels via Heterotrimeric G-proteins in Neurons
- Inhibitory Effects of Bifemelane on Brain Ca^ Channel Subtypes Expressed in Xenopus Oocytes
- Inhibition by [Arg8]-Vasopressin of Long Term Potentiation in Guinea Pig Hippocampal Slice
- Involvement of M_2 receptor in an enhancement of long-term potentiation by carbachol in Schaffer collateral-CA1 synapses of hippocampal slices
- NADPH Oxidase Isoforms and Anti-hypertensive Effects of Atorvastatin Demonstrated in Two Animal Models
- The Activity of Aldose Reductase Is Elevated in Diabetic Mouse Heart
- Mechanisms of Chronic Nicotine Treatment-Induced Enhancement of the Sensitivity of Cortical Neurons to the Neuroprotective Effect of Donepezil in Cortical Neurons
- Negative Relationship Between Morphine Analgesia and P-Glycoprotein Expression Levels in the Brain
- Neuronal Nitric Oxide Synthase Is Crucial for Ganglion Cell Death in Rat Retinal Explant Cultures
- Na^+/Ca^ Exchanger Inhibitors Inhibit Neurite Outgrowth in PC12 Cells
- Glutathione Biosynthesis via Activation of the Nuclear Factor E2-Related Factor 2 (Nrf2)-Antioxidant-Response Element (ARE) Pathway Is Essential for Neuroprotective Effects of Sulforaphane and 6-(Methylsulfinyl) Hexyl Isothiocyanate
- In Vivo Brain Oxidative Stress Model Induced by Microinjection of Sodium Nitroprusside in Mice
- Glucocorticoids Decrease Astrocyte Numbers by Reducing Glucocorticoid Receptor Expression In Vitro and In Vivo
- In Vivo Brain Oxidative Stress Model Induced by Microinjection of Sodium Nitroprusside in Mice
- Protective Effect of Luteolin on an Oxidative-Stress Model Induced by Microinjection of Sodium Nitroprusside in Mice
- Involvement of Peripheral Mechanism in the Verapamil-Induced Potentiation of Morphine Analgesia in Mice
- HMGB1 as a Potential Therapeutic Target for Neuropathic Pain
- Possible Involvement of Endogenous Opioid System Located Downstream of α7 Nicotinic Acetylcholine Receptor in Mice With Physical Dependence on Nicotine