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Department Of Oncology (c6) Graduate School Of Medicine Osaka University | 論文
- A mechanism underlying STAT4-mediated up-regulation of IFN-γ induction in TCR-triggered T cells
- Reversible CD8 expression induced by common cytokine receptor γchain-dependent cytokines in a cloned CD4^+ T_h1 cell line
- Requirement of IFN-γ in IL-12 production induced by collaboration between V_α14^+ NKT cells and antigen-presenting cells
- CD28 co-stimulatory signals induce IL-2 receptor expression on antigen-stimulated virgin T cells by an IL-2-independent mechanism
- B7.2-Ig fusion proteins enhance IL-4-dependent differentiation of tumor-sensitized CD8^+ cytotoxic T lymphocyte precursors
- The capacity of the natural ligands for CD28 to drive IL-4 expression in naive and antigen-primed CD4^+ and CD8^+ T cells
- Histiocytoid breast carcinoma : Solid variant of invasive lobular carcinoma with decreased expression of both E-cadherin and CD44 epithelial variant
- Differential IL-12 responsiveness of T cells but not of NK cells from tumor-bearing mice in IL-12-respomsive versus-unresponsive tumor models
- MOLECULAR MECHANISMS UNDERLYING T CELL MIGRATION RESPONSIBLE FOR IL-12-INDUCED TUMOR REGRESSION
- Molecular mechanisms underlying IFN-γ-mediated tumor growth inhibition induced during tumor immunotherapy with rIL-12
- B cell-mediated down-regulation of IFN-γ and IL-12 Production induced during antitumor immune responses in the tumorbearing state
- Association of a tetraspanin CD9 with CD5 on the T cell surface: role of particular transmembrane domains in the association
- Inhibition of Growth and Metastasis of Ovarian Carcinoma by Administering a Drug Capable of Interfering with Vascular Endothelial Growth Factor Activity
- A Defect in Cell-to-cell Adhesion via Integrin-Fibronectin Interactions in a Highly Metastatic Tumor Cell Line
- Do Structural Changes of T Cell Receptor Complex Occur in Tumor-bearing State?
- Systemic administration of rIL-12 induces complete tumor regression and protective immunity: response is correlated with a striking reversal of suppressed IFN-γ production by anti-tumor T cells
- Cyclosporin A inhibits late steps of T lymphocyte activation after transmembrane signaling.