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Department Of Diagnostic Medicine Graduate School Of Medical And Pharmaceutical Sciences Kumamoto Un | 論文
- A Case of Familial Amyloid Polyneuropathy due to Phe33Val TTR with Vitreous Involvement as the Initial Manifestation
- Severe congestive heart failure with cardiac liver cirrhosis 10 years after orthotopic liver transplantation for familial amyloidotic polyneuropathy
- Inhibitory Effect of α-Tocopherol on Methylmercury-Induced Oxidative Steress
- Clinical Investigation of the Lesions Responsible for Sensory Disturbance in Minamata Disease
- Distribution of Dystrophin and Dystrophin-Associated Protein 43DAG(β-dystroglycan) in the Central Nervous System of Normal Controls and Patients with Duchenne Muscular Dystrophy
- Nitric Oxide Synthase Activity in Human Lung Cancer
- Reuse of a Japanese Familial Amyloidotic Polyneuropathy Patient's Liver for a Cancer Patient: The Domino Liver Transplantation Procedure
- Midkine in plasma as a novel breast cancer marker
- Significant Correlation of Nitric Oxide Synthase Activity and p53 Gene Mutation in Stage I Lung Adenocarcinoma
- Evaluation of Myocardial Changes in Familial Amyloid Polyneuropathy after Liver Transplantation
- Change in the Age of Onset in Patients with Familial Amyloidotic Polyneuropathy Type I
- Familial amyloidotic ployneuropathy(ATTR Val30Met) with widespread cerebra lamyloid angiopathy and lethal cerebral hemorrhage
- Late onset type I familial amyloidotic polyneuropathy : Presentation of three autopsy cases in comparison with 19 autopsy cases of the ordinary type
- Nodular Fasciitis in the Buccal Mucosa : A Case Report
- Reduced expression of ubiquitin ligase FBXW7 mRNA is associated with poor prognosis in breast cancer patients
- Multicenter evaluation of prototype real-time PCR assays for Epstein-Barr virus and cytomegalovirus DNA in whole blood samples from transplant recipients
- Manifestations of Transthyretin-Related Familial Amyloidotic Polyneuropathy : Long-Term Follow-Up of Japanese Patients After Liver Transplantation
- Renal amyloidosis caused by apolipoprotein A-II without a genetic mutation in the coding sequence