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千葉大学 循環病態医科学 | 論文
- The Japanese Society of Hypertension Committee for Guidelines for the Management of Hypertension
- 27) Great Cardiac Veinでのカテーテルアブレーションにより根治しえた左室流出路起源心室頻拍の1例(第114回日本循環器学会北陸地方会)
- DPE-008 Bone Marrow Derived Mononuclear Cells Improve the Cardiac Function of Dilated Cardiomyopathy Models via Gr-1 Positive Cells-secreted Factors(DPE02,Heart Failure, Basic (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of The
- PE-424 Bone Marrow Derived Monomuclear Cells Transiently Improve the Cardiac Function of Dilated Cardiomyophthy Models via Gr-1 Positive cells-secreted Factors(Heart failure, basic(04)(M),Poster Session(English),The 72nd Annual Scientific Meeting of the J
- PE-556 Novel Method for Displaying Left Ventricular Function and Dyssynchrony Using Doppler Tissue Imaging : Its Application in Acute Pacing Study(PE093,Echo/Doppler (Others) (I),Poster Session (English),The 73rd Annual Scientific Meeting of the Japanese
- Left Atrial Dyssynchrony in Patients With Nonobstructive Hypertrophic Cardiomyopathy Evaluated by Myocardial Strain Imaging
- OE-164 Atg7 is Essential for Starvation-induced And Constitutive Autophagy in the Hearts(Molecular biology, myocardium (M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-020 Molecular and Structural Basis for Mechanical Stress-induced Activation of AT Receptor(Cardiovascular pharmacology, basic/clinical(01)(H),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- FRS-009 Crucial roles of PDK1 in the regulation of β-adrenergic response and cell survival in the hearts(Novel Pathophysiological Mechanisms of Heart Failure(M),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Socie
- Angiotensin II Type 1a Receptor Signals are Involved in the Progression of Heart Failure in MLP-Deficient Mice
- OJ-256 Erythropoietin Prevents Apoptotic Cell Death Through Erythropoietin Receptor-mediated Cytoprotective Signaling Pathways(Growth factor/Cytokine-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- OE-056 Erythropoietin Prevents LV Remodeling after Myocardial Infarction through Erythropoietin Receptor-Induced Signaling Pathways in the Heart(Acute myocardial infarction, basic-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- 1 p53-induced Inhibition of Angiogenesls Causes Cardiac Dysfunction during Pressure Overload(The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- PE-178 Different Aspects of AT1 Receptor Blockers in Showing Inverse Agonist Activity(Cardiac hypertrophy, basic/clinical-3 (M) PE30,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- PE-074 Erythropoietin Directly Activates Intracellular Signaling Pathways and Prevents Apoptotic Cell Death in Cardiomyocytes(Cardiovascular pharmacology, basic/clinical-3 (H) PE13,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of
- OJ-316 G-CSF Directly Inhibits Myocardial Ischemia-Reperfusion Injury through Akt-eNOS Pathway(Growth factor/Cytokine-1 (H) OJ53,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- OJ-242 Erythropoietin Has an Ischemic Postconditioning-like Effect on Myocardium through a Direct Action on Cardiomyocytes(Myocardial ischemia-reperfusion, basic/clinical-2 (M) OJ41,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeti
- 1 Actions on Angiotensin II Receptor by Angiotensin II, Mechanical Stress, And Inverse Agonists(Molecular Mechanism of Hypertensive Heart Failure,Symposium 5 (SY5) (H),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- G-CSF Prevents Cardiac Remodeling after Myocardial Infarction by Activating Jak/STAT in Cardiomyocytes(Vascular Regeneration (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Amelioration of Hypertensive Heart Failure by Amlodipine May Occur via Antioxidative Effects
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