Sakai Satoshi | Cardiovascular Division Graduate School Of Comprehensive Human Science University Of Tsukuba

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著作論文

PE-036 Contrast Medium-Induced Renal Damage is Tightly Associated with Endothelial Injury in Patients(PE006,Kidney/Renal Circulation/CKD 1 (H),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
DPJ-048 Can Intravascular Optical Cohenrence Tomography Detect Plaque Erosion? : Some New Findings in Rabbit Models(DPJ08,Intravascular Imagings (I),Digital Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
DPJ-021 Commercially Available Serum N-terminal proBNP Level Accurately Differentiates Presence of Cardiac Morphological and Hemodynamic Abnormalities Compared with Brain Natriuretic Peptide(DPJ04,Heart Failure (Diagnosis) (M),Digital Poster Session (Japa
OJ-045 Altered Characteristics of Endothelial Cells in Patients with Pulmonary Arterial Hypertension Estimated by Endothelial Cell-Derived Microparticles(OJ08,Pulmonary Circulation (H),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The
OE-336 Optical Coherence Tomographic Findings of Macrophage Accumulation in Atherosclerotic Lesions(OE57,Intravascular Imagings (I),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
PE-369 The Histological Findings of Vascular Response to Drug Eluting Stents(PE062,Atherosclerosis (Clinical/Treatment) (IHD),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
PE-233 Validation of Left Ventricular Volumetric Data by 3 Dimensional Speckle Tracking Imaging with Cardiac Magnetic Resonance Imaging(PE039,Echo/Doppler (Others) (I),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation
Poor Survival of Cardiomyopathic Hamsters with CHF Is Greatly Improved by Combined Treatment with Endothelin (ET)-A Receptor Antagonist and ACE Inhibitor
In vivo Direct Nitric Oxide (NO) Measurement in the Rat Abdominal Aorta Using Novel Clinically Practicable NO Sensor-tipped Catheter(Atherosclerosis, Basic 6 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-332 Left Atrial Volumes Assessed by Echocardiography Compared to Magnetic Resonance Imaging(Echo/Doppler-12, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OJ-065 Inhibitory Effect of Fluvastatin on Cardiac hypertrophy is Partly Attributed to Interference of JNK and Pin-1 Activation(Cardiovascular pharmacology, basic/clinical(02)(H),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japane
PJ-636 Regulation of RNA Polymerase II (Pol II)-Driven Gene Expression and Cardiomyocyte Size by C-Terminal Domain Phosphatase of Pol II(PJ107,Cardiomyopathy/Hypertrophy (Basic, Clinical) (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of
PJ-618 The PPAR-α Activator Fenofibrate Inhibits Myocardial Fibrosis with Improving Diastolic Function in DOCA-salt Hypertensive Rats, Partly via Suppressing Endothelin-1 Expression(Cardiac Hypertrophy, Basic and Clinical 2 (M) : PJ104)(Poster Session (Ja
Decreases in Exercise Capacity, Myocardial Energy Metabolism, and Survival in Rats with CHF Are Greatly Improved by Endothelin-A Receptor Antagonist
Cardiac Hypertrophy by Hypertension and Exercise Training Exhibits Different Gene Expression of Enzymes in Energy Metabolism
Fenofibrate, one of peroxisome proliferator-activated receptor α (PPAR-α) activators, inhibits cardiac fibrosis by depression of both fibroblast proliferation and collagenesis
Stimulation of Peroxisome Proliferator-Activated Receptor-α (PPAR-α) Attenuates Endothelin-1-Induced Cardiac Hypertrophy Partly via Inhibition of c-Jun NH_2 Terminal Kinase (JNK) Pathway
Prediction of Chronic Renal Insufficiency After Coronary Angiography by an Early Increase in Oxidative Stress and Decrease in Glomerular Filtration Rate
Combination of endothelin-A (ET_A) receptor inhibitor and prostacyclin (PGI_2) analogue is preferable to each drug alone for pulmonary hypertension (PH)
A PPAR-α Activator Inhibits Cardiac Fibrosis by Inhibiting of Fibroblast Proliferation and Collagenesis in Cultured Fibroblasts and DOCA-Salt Hypertensive Rats

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