Sakai Satoshi | Cardiovascular Division, Graduate School of Comprehensive Human Sciences University of Tsukuba

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著作論文

DPJ-048 Can Intravascular Optical Cohenrence Tomography Detect Plaque Erosion? : Some New Findings in Rabbit Models(DPJ08,Intravascular Imagings (I),Digital Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
DPJ-021 Commercially Available Serum N-terminal proBNP Level Accurately Differentiates Presence of Cardiac Morphological and Hemodynamic Abnormalities Compared with Brain Natriuretic Peptide(DPJ04,Heart Failure (Diagnosis) (M),Digital Poster Session (Japa
OJ-045 Altered Characteristics of Endothelial Cells in Patients with Pulmonary Arterial Hypertension Estimated by Endothelial Cell-Derived Microparticles(OJ08,Pulmonary Circulation (H),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The
OE-336 Optical Coherence Tomographic Findings of Macrophage Accumulation in Atherosclerotic Lesions(OE57,Intravascular Imagings (I),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
OE-207 Elevated Blood Pressure in Resting Daytime-Phase in A170-Knockout Mice, a Newly Established Obese Model(OE35,Metabolic Syndrome (H),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
OE-290 A170 Deficiency Causes Enhanced Vascular Remodeling and Vascular Smooth Muscle Cell Proliferation(Vascular smooth muscle (IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
Poor Survival of Cardiomyopathic Hamsters with CHF Is Greatly Improved by Combined Treatment with Endothelin (ET)-A Receptor Antagonist and ACE Inhibitor
IS004 Cloning of hamster prepro-endothelin-1 cDNA and the pathophysiological roles of myocardial endothelin-1 in the failing heart due to cardiomyopathy in hamsters
IS097 Chronic treatment with endothelin inhibitor greatly improves survival in hamsters with heart failure due to dilated cardiomyopathy : increased expression of endothelin-1 in failing heart
PE-332 Left Atrial Volumes Assessed by Echocardiography Compared to Magnetic Resonance Imaging(Echo/Doppler-12, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
P627 EXPRESSION OF ENDOTHELIN RECEPTOR SUBTYPBS AND ANGIOTENSIN RECEPTOR SUBTYPES IN THE HEART OF RATS WITH CHRONIC HEART FAILURE
OJ-065 Inhibitory Effect of Fluvastatin on Cardiac hypertrophy is Partly Attributed to Interference of JNK and Pin-1 Activation(Cardiovascular pharmacology, basic/clinical(02)(H),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japane
PJ-636 Regulation of RNA Polymerase II (Pol II)-Driven Gene Expression and Cardiomyocyte Size by C-Terminal Domain Phosphatase of Pol II(PJ107,Cardiomyopathy/Hypertrophy (Basic, Clinical) (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of
PJ-618 The PPAR-α Activator Fenofibrate Inhibits Myocardial Fibrosis with Improving Diastolic Function in DOCA-salt Hypertensive Rats, Partly via Suppressing Endothelin-1 Expression(Cardiac Hypertrophy, Basic and Clinical 2 (M) : PJ104)(Poster Session (Ja
Decreases in Exercise Capacity, Myocardial Energy Metabolism, and Survival in Rats with CHF Are Greatly Improved by Endothelin-A Receptor Antagonist
0894 EXPRESSION OF ENDOTHELIN RECEPTOR MESSENGER RNA IN THE CONGESTIVE LUNG IN RATS WITH CHRONIC HEART FAILURE
Cardiac Hypertrophy by Hypertension and Exercise Training Exhibits Different Gene Expression of Enzymes in Energy Metabolism
Fenofibrate, one of peroxisome proliferator-activated receptor α (PPAR-α) activators, inhibits cardiac fibrosis by depression of both fibroblast proliferation and collagenesis
Stimulation of Peroxisome Proliferator-Activated Receptor-α (PPAR-α) Attenuates Endothelin-1-Induced Cardiac Hypertrophy Partly via Inhibition of c-Jun NH_2 Terminal Kinase (JNK) Pathway
Prediction of Chronic Renal Insufficiency After Coronary Angiography by an Early Increase in Oxidative Stress and Decrease in Glomerular Filtration Rate
Combination of endothelin-A (ET_A) receptor inhibitor and prostacyclin (PGI_2) analogue is preferable to each drug alone for pulmonary hypertension (PH)
Corresponding distributions of increased endothelin-B receptor expression and increased endothelin-1 expression in the aorta of apolipoprotein E-deficient mice with advanced atherosclerosis

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