Tobe Kazuyuki | Department Of Metabolic Diseases

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著作論文

Inhibition of Tumor Progression Locus 2 Protein Kinase Decreases Lipopolysaccharide-Induced Tumor Necrosis Factor α Production Due to the Inhibition of the Tip-Associated Protein Induction in RAW264.7 Cells
FRS-060 SUMOylation of KLF5 is a key molecular switch of transcriptional regulatory programs of lipid metabolism(New Insights into Metabolic Syndrome(02)(H),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PE-411 KLF5 Controls Metabolic Stress Response in Metabolic Syndrome and Cardiovascular Disease(Diabetes/Obesity/Metabolic syndrome-05, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Lack of Insulin Receptor Substrate-2 Is a Link between Insulin Resistance and an Increased Intima of the Vessel in Mice
Vascular Intima Formation in Insulin Receptor Substrate-2 Deficient Mice
Biphasic Activation of Two Mitogen-activated Protein Kinases during the Cell Cycle in Mammalian Cells.
Mitogen-induced Tyrosine-phosphorylated 41- and 43-kDA Proteins Are Family Members of Extracellular Signal-regulated Kinases/ Microtubule-associated Protein 2 Kinases.
PE-324 Adiponectin May Effect on Angiotensin II Stimulations(Vascular smooth muscles-2 (H) PE54,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Adiponection Inhibits the Transcriptional Circuitry Controlling Vascular Remodeling in Smooth Muscle Cells(Vascular Smooth Muscle 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Signal Transduction Mechanism of Insulin and Insulin-Like Growth Factor-1
Kruppel-like Transcription Factor KLF5 is a Key Regulator Governing Adipocyte Differentiation(Metabolic Syndrome (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Antibody-mediated Insulin Resistance Treated by Cessation of Insulin Administration
OJ-046 KLF5 Plays Pivotal Roles in Pathogenesis of both Metabolic Syndrome and Vascular Disease(Diabetes/Obesity/Metabolic syndrome-3 (H) OJ8,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OE-316 Kruppel-like family of transcription factor 5 is required for adipocyte differentiation(Diabetes/Obesity 2 (H) : OE40)(Oral Presentation (English))
The Mechanism of Insulin-induced Signal Transduction Mediated by the Insulin Receptor Substrate Family
0863 ANALYSIS OF FOCAL ADHESION RELATED TYROSINE KINASE IN CARDIAC MYOCYTE STIMULATED BY VASCULAR ENDOTHELIAL GROWTH FACTOR (VEGF)
MOLECULAR ANALYSIS OF CARDIAC HYPERTROPHY BY MECHANICAL STRESS:INVOLVEMENT OF STRETCH-INDUCED PROTEIN KINASE CASCADE
C57BL/6JJclを遺伝的背景に持つIRS2欠損マウスの効率的な繁殖方法の検討
Phenotypes of IRS-2 Deficient Mice Produced by Reproductive Technology are Stable
Maturity-onset Diabetes of the Young Resulting from a Novel Mutation in the HNF-4α Gene
Respiratory involvement in IgG4-related Mikulicz's disease

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