Ohtani Kisyou | Department Of Cardiovascular Medicine Graduate School Of Medicine Sciences Kyushu University

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著作論文

Stromal Cell-Derived Factor-1α Gene Transfer Induces Therapeutic Angiogenesis by Recruiting Endothelial Progenitor Cells via the VEGF/eNOS Pathway
Identification of a novel gene involved in the pathogenesis of cardiac hypertrophy in rats
HMG-CoA Reductase Inhibitor Limits Angiotensin II-Induced Progression and Destabilization of Established Atherosclerosis in ApoE-Knockout Mice through Its Cholesterol-Lowering Independent Actions
Anti-Monocyte Chemoattractant Protein-1 Gene Therapy Limits Progression and Destabilization of Established Atherosclerosis in Apolipoprotein E-Knockout Mice
Anti-monocyte chemoattractant protein-1 gene therapy inhibits neointimal hyperplasia after balloon angioplasty in rats and monkeys
Monocyte Chemoattractant Protein-1 is Vital in Angiotensin II-induced Progression and Destabilization of Established Atherosclerosis in Apolipoprotein E-Knockout Mice
Anti-inflammation Strategy Via Local Gene Transfer with Geneeluting Stents Attenuates in-stent Restenosis in Hypercholesterolemic Rabbits and Monkeys(Coronary Revascularization, PTCA/Stent/DCA/Rotablator/New Device 16 (IHD), The 69th Annual Scientific Mee
OE-123 Blockade or Abrogation of Angiotensin II AT1 Receptor Suppressed Accelerated Diabetic Atherosclerosis in Streptozotosin-Induced Diabetic Apolipoprotein E-Deficient Mice(Atherosclerosis, Basic 1 (IHD) : OE14)(Oral Presentation (English))
OE-120 Cholesterol-lowering independent regression and stabilization of atherosclerotic lesions by pravastatin in nonhuman primates(Atherosclerosis, Basic 1 (IHD) : OE14)(Oral Presentation (English))
Upregulation of Monocyte Chemoattractant Protein (MCP)-1 Receptor in Circulating Monocytes Contributes to Angiotensin II : Induced Arteriosclerosis/Atherosclerosis
Enhanced Expression of MCP-1 Receptor in Circulating Monocytes in Hypertensive Patients through Angiotensin II Type 1 Receptor
Angiotensin II AT1 Receptor-Mediated Enhanced RAGE Signals are Responsible for the Mechanism of Accerelated Atherosclerosis by Diabetes Mellitus(Atherosclerosis, Basic 10 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Blockade of Vascular Endothelial Growth Factor Attenuates Experimental Restenosis after Intraluminal Injury
2 Stent-Based Local Delivery of Nuclear Factor-k B Decoy Attenuates In-Stent Restenosis in Hypercholesterolemic Rabbits(Young Investigator's Award Finalists Lecture (YIA),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
A Third-generation Long-acting Dihydropyridine Calcium Channel Blocker, Azelnidipine Attenuates Stent-associated Neointimal Formation in Hypercholesterolemic Non-human Primates (Restenosis after Angioplasty, Basic/Clinical 6 (IHD), The 69th Annual Scienti
A Third-generation Long-acting Dihydropyridine Calcium Channel Blocker, Azelnidipine Attenuates Atherosclerosis Through Antioxidant Actions in Hypercholesterolemic Mice and Monkeys(Atherosclerosis, Basic 5 (IHD), The 69th Annual Scientific Meeting of the
Catheter-based Adenovirus-mediated Local Gene Transfer of Anti-inflammation Factor Attenuates In-stent Neointimal Formation in Non-human Primates(Restenosis after Angioplasty, Basic/Clinical 5 (IHD), The 69th Annual Scientific Meeting of the Japanese Circ
OE-121 Absence of monocyte chemoattractant protein-1 receptor CCR2 on monocytes supresses angiotensin II-induced progression of atherosclerosis in ApoE-deficient mice(Atherosclerosis, Basic 1 (IHD) : OE14)(Oral Presentation (English))
FRS-166 Stent-based delivery of nuclear factor-κB(NF-κB) attenuates in-stent restenosis in hypercholesterolemic rabbits(Coronary Heart Disease : Basic Science (IHD) : FRS20)(Featured Research Session (English))
Anti-Monocyte Chemoattractant Protein-1 Therapy Reduces Experimental In-Stent Restenosis in Hypercholesterolemic Rabbits
A gene therapy targeting early growth response factor-1 by "decoy" strategy inhibits neointinal hyperplasia in hypercholesterolemic rabbits
Pioglitazone prevents Coronary Arteriosclerosis through inhibition of inflammation by downregulation of monocyte CCR2 in Rat

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