Horai Reiko | National Human Genome Research Institute National Institutes Of Health

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著作論文

P6-08 Possible role of inflammatory cytokine in liver injury caused by cocaine(LIVER AND DIGESTIVE SYSTEM/CIRCULATORY SYSTEM-1/DEVELOPMENT AND REPRODUCTION)(GENERAL SESSION BY POSTER PRESENTATION)(Proceedings of the 31st Annual Meeting)
Possible role of inflammatory cytokines in liver injury caused by cocaine (LIVER/DIGESTIVE SYSTEM) (GENERAL SESSION BY POSTER PRESENTATION) (Proceedings of the 30th Annual Meeting)
Suppression of oxidative neuronal damage after transient middle cerebral artery occlusion in mice lacking interleukin-1
O11-04 A role of inflammatory cytokine in CYP down-regulation caused by BCG or LPS
Increased Expression of Interleukin-1β in Mouse Hippocampus after Global Cerebral Ischemia
IL-1α, but not IL-1β, is required for contact-allergen-specific T cell activation during the sensitization phase in contact hypersensitivity
Interleukin 1 Receptor Antagonist Is Important in the Suppression of Neointima Formation after Injury
3 Deficiency of lnterleukin-1 Receptor Antagonist Increases the Atherosclerosis in Apolipoprotein E-Deficient Mice(Young Investigator's Award Finalists Lectures (YIA))(Special Program)
Abnormal T cell activation caused by the imbalance of the IL-1/IL-1R antagonist system is responsible for the development of experimental autoimmune encephalomyelitis
IL-1 is required for allergen-specific T_h2 cell activation and the development of airway hypersensitivity response
IL-1-induced tumor necrosis factor-α elicits inflammatory cell infiltration in the skin by inducing IFN-γ-inducible protein 10 in the elicitation phase of the contact hypersensitivity response
O-24 HEME OXYGENASE GENE EXPRESSION BY LIPOPOLYSACCHARIDE IN IL-1 ALPHA/BETA KNOCK OUT MOUSE.(Proceedings of the 27th Annual Meeting)
22C-11-1 Allergic Contact Dermatitis in IL-1 α/β Doubly Deficient Mice.

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