Ohara Tomoki | Center For Advanced Medical Engineering And Informatics Osaka University

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著作論文

PJ-084 Diastolic function is impaired at the noninfarcted region in patients with LV remodeling after myocardial infarction(Echo/Doppler(11)(I),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
Is Amount of Thrombus At Culprit Lesion Associated With Microvascular Damage in Patients With Acute Myocardial Infarction?
Coronary Remodeling as a Determinant for the Amount of Thrombus Formed at the Culprit Lesions of Acute Myocardial Infarction
Sustained elevation of brain natriuretic peptide (BNP) concentrations in patients with left ventricular remodeling after myocardial infarction
Smoking Status on Thrombus and Plaque in The Culprit Vessel in Patients with Acute Myocardial Infarction : Angioscopic Findings
Advanced age increased the occurrence of heart failure in patients with acute myocardial infarction due to increased ischemic risk area
Correlation Between Intima-Media Thickening of Carotid Arteries and Intra-Coronary Yellow Plaques
Abnormal Myocardial Free Fatty Acid Utilization Deteriorates With Morphological Changes in the Hypertensive Heart
-0721-DOES REDISTRIBUTION IN EXERCISE THALLIUM-201 SINGLE PHOTON EMISSION COMPUTERIZED TOMOGRAPHY REPRESENT RESIDUAL ISCHEMIA IN OLD MYOCARDIAL INFARCTION?
-0593-HIBERNATING MYOCARDIUM IN INFARCTED AREA : ASSESSMENT AFTER PTCA AT CHRONIC STAGE
-0221-THE EFFECT OF ATRIAL CONTRACTION AND SYNCHRONEITY OF CARDIAC CONTRACTION ON VENTRICULO-ARTERIAL COUPLING AND MECHANICAL ENERGY EFFICIENOY
-0828-BIPHASIC RELEASE OF CARDIAC MYOSIN LIGHT CHAIN II IN PATIENTS OF ACUTE MYOCARDIAL INFARCTION WITH SUCCESSFUL RECANALIZATION : THE 54th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY
PE-284 Angiotensin Converting Enzyme Inhibitor And Angiotensin II Type1 Receptor Antagonist Differently Modulate Extracellular Matrix Regulatory System in Diastolic Heart Failure(Heart Failure, Clinical 10 (M) : PE49)(Poster Session (English))
PE-182 Angiotensin II type1 Receptor Blocker Suppresses ROS Generation and Inflammatory Changes and Prevents Overt Diastolic Heart Failure in Hypertensive Hearts(Infection/Inflammation/Immunity 2 (H) : PE31)(Poster Session (English))
No-Reflow Following Dilatation of a Coronary Lesion With a Large Lipid Core
Thrombectomy with Direct PTCA for Acute Coronary Syndrome(ACS) Improves Regional Left Ventricular Systolic Function
Clinical experience of self expandable RADIUS stent : Initial and late term results
Exploration for Optimal Strategy to Acute Coronary Syndrome(ACS)-recent clinical outcomes of percutaneous coronary intervention(PCI) in our hospital
Intravascular Ultrasound Imaging of unstable plaque can predicts angiological `no-reflow` during coronary intervention
Asymmetrical Dimethyl Arginine (ADMA) is a Novel Marker of Coronary Stent Restenosis
Prediction of No-Reflow Phenomenon After Successful Percutaneous Coronary Intervention in Patients With Acute Myocardial Infarction : Intravascular Ultrasound Findings
Disappearance of Intima Over the Stent and Ulcer Formation After Intracoronary Radiation for ln-Stent Restenosis : Angioscopic Findings in Human Coronary Arteries
Angioscopic Findings of Neointima after Intracoronary Radiation Therapy in Human
Intracoronary Plasma Level of Matrix Metalloproteinase(MMP)-9 Elevates after Interventional Procedure in Acute Myocardial Infarction (AMI)
Long Term Outcome of Vascular Brachytherapy (VBT) Using P32 for In-Stent Restenosis
Elevation of Plasma Matrix Metalloproteinase-9 in the Culprit Coronary Artery in Patients With Acute Myocardial Infarction : Clinical Evidence From Distal Protection
PE-441 A new strategy for angioplasty of the superficial femoral artery : bidirectional brachio-popliteal approach(Peripheral Circulation/Vascular Disease 4(H) : PE75)(Poster Session (English))
PE-420 Regression of Neointima Following Intracoronary Radiation Therapy : Angioscopic Finding in Patients(Intravascular Endoscopy/Intravascular Ultrasound 6 (I) : PE71)(Poster Session (English))
PE-340 Vulnerable Plaque can Be the Predominant Source of lnterleukin-6 but not of C-Reactive Protein in Patients With Acute Myocardial Infarction(Acute Myocardial Infarction, Clinical (Pathophysiology) 7 (IHD) : PE58)(Poster Session (English))
PE-246 Addition of Distal Embolic Protection Device to Primary Angioplasty for Acute Myocardial Infarction Does not Improve Myocardial Salvage(Nuclear Cardiology 9 (I) : PE42)(Poster Session (English))
PE-241 Thrombectomy Prior to Angioplasty Has Potential to Salvage Myocardium at Risk in Acute Myocardial Infarction(Nuclear Cardiology 8 (I) : PE41)(Poster Session (English))
PE-193 Positive Isovolumic Relaxation Velocity Detected by a Simplified Tissue Doppler Mapping Technique Suggests Coronary Artery Stenosis of Normally Contracting Heart(Echo/Doppler 9 (I) : PE33)(Poster Session (English))
PE-155 Distal Protection Assisted Acute Infarct Intervention Can not Prevent Left Ventricular Remodeling(Acute Myocardial Infarction, Clinical (Diagnosis/Treatment) 7 (IHD) : PE27)(Poster Session (English))
OE-129 Thrombotic Occlusive Coronary Lesions may Be the Source of Plasma Matrix Metalloproteinase-9 in Patients With Acute Myocardial Infarction(Coronary Circulation, Basic/Clinical 2 (IHD) : OE15)(Oral Presentation (English))
4 Current problems in the treatment of acute coronary syndrome : distal embolization and inadequate reperfusion following PCl(Plenary Session 1 (PL1) (IHD) : ACS : Prevention to Treatment)(Special Program)
-0859-ULTRASTRUCURAL STUDY OF THE SITE OF RESTENOSIS AFTER DIRECT PTCA AND PTCR & A : DIFFERENCE BETWEEN DIRECT PTCA AND PTCR & A : THE 54th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY
-0584-SILENT ISCHEMIA DURING CORONARY OCCLUSION PRODUCED BY TWICE BALLOON INFLATIONS : RELATION TO LACTATE METABOLISM
-0055-ENHANCED ADENOSINE RELEASE AS A POSSIBLE MECHANISM OF WARM UP PHENOMENON
Formation of Intra-Coronary Yellow Plaque Is Associated with Deterioration of Nitric Oxide Bioavailability
Myocardial Stiffening Is Not Due to Myocardial Hypertrophy but to Myocardial Fibrosis in Human Left Ventricular Hypertrophy
Amount of Thrombus at Culprit Lesion of Acute Myocardial Infarction Is Dependent on Angioscoopically Determined Coronary Atherosclerosis
Gradual Recovery of Thallium Uptake at the Infarct Segement in Patients with Infarct Artery Patency after Myocardial Infarction
Coverage of Culprit Plaque by Stent and the Rate of Restenosis
PCI for ACS Causes Distal Microembolization of Lipid-Rich Material in Most Cases, Pathological Study Using Distal Protection Device (Percuserge)
Troponin T and Coronary Plaque Morphology by Angioscopic Evaluation
Does the Thrombus Burden Cause No-Reflow During Angioplasty? : A Histologic Analysis of Aspirates from Infarct-Related Arteries Before and After Intervention
Angioscopically Evaluated Atherosclerosis Is Associated With the Acute Coronary Events During 5 Years Follow-up After Acute Myocardial Infarction

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