Kawamura Teruhisa | Department of Cardiology, Graduate School of Medicine, Kyoto University

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著作論文

OE-363 Activation of cardiac aldosterone system induces association of mineralocorticoid receptor with transcriptional coactivator p300 during hypertrophic responses in cardiac myocytes(Cardiac Hypertrophy, Basic and Clinical 1 (M) : OE46)(Oral Presentati
OE-362 Lys-CoA, a Specific Inhibitor of p300-Mediated Acetylation, Represses Hypertrophic Responses in Cardiac Myocytes(Cardiac Hypertrophy, Basic and Clinical 1 (M) : OE46)(Oral Presentation (English))
A Specific Inhibitor of Histone Deacetylase, Trichostatin A, Promotes Differentiation of Embryonic Stem Cells into Cardiac Myocytes
Overexpression of P300 Promotes Left Ventricular Endothelin-1 Expression and Remodeling Following Myocardial Infarction in Adult Mice in Vivo
A Transcriptional Coactivator P300 Associates with Calcineurin-Dependent Signaling during the Transcription of Bcl-2 Gene in Cardiac Myocyte
Cardiac Nuclear Hyper-Acetylation by Inhibiting Histone Deacetylase Facilitates Left Ventricular Remodeling Following Myocardial Infarction In Adult Mice in Vivo
Overexpression of p300 Prevents the Development of Doxorubicin-induced Cardiomyopathy in Adult Mice in vivo
A p300 Protein Controls the Transcription of Bcl-2 Gene and Promotes Myocardial Cell Survival in Association with NFATc and GATA-4
A Ligand for Peroxisome Proliferator-Activated Receptor Inhibits Anti-apoptotic Effects of Endothelin-1 on Cardiac Myocytes
Administration of Recombinant Human Granulocyte-Colony Stimulating Factor Deteriorates Doxorubicin-lnduced Cardiomyopathy in Adult Mice in Vivo

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