Kawakami Rika | Department Of Medicine And Clinical Science Kyoto University Graduate School Of Medicine

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著作論文

NRSF Is Involved in Molecular Pathways for Sudden Death Associated with Heart Failure
IS003 Differing role of Extracellular signal-regulated kinase in endothelin-1-induced and cardiotrophin-1-induced hypertrophic signals in cultured cardiac myocytes
Genetic Disruption of Angiotensin II Type 1a Receptor Improves Long-Term Survival of Mice With Chronic Severe Aortic Regurgitation
In-Vivo Electrophysiological Study in Dominant-Negative NRSF Transgenic Mice : Ventricular Arrhythmia Vulnerability and Sudden Cardiac Death
Angiotensin II-Induced Ventricular Hypertrophy and Extracellular Signal-Regulated Kinase Activation Are Suppressed in Mice Overexpressing Brain Natriuretic Peptide in Circulation
OJ-021 Blockade of T-type Calcium Channel Reduced the Arrythmogenicity in the Dominant Negative NRSF Transgenic Mice(Heart failure, basic-2 (M) OJ4,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Soc
SOCS1/JAB Likely Mediates the Protective Effect of Cardiotrophin-1 Against Lipopolysaccharide-Induced Left Ventricular Dysfunction In Vivo
Overexpression of Brain Natriuretic Paptide Attenuates Cardiac Remodeling after Myocardial Infarction in Mice(Old Myocardial Infarction/Remodeling 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PJ-622 Significance of a novel interaction between NRSF and classII HDAC involved in the induction of ANP gene expression by Endothelin-1(Cardiac Hypertrophy, Basic and Clinical 2 (M) : PJ104)(Poster Session (Japanese))
OJ-481 Beneficial Effects of Brain Natriuretic Peptide Overexpression in Cardiac Remodeling after Myocardial Infarction in Mice(Old Myocardial Infarction/Remodeling 1 (IHD) : OJ59)(Oral Presentation (Japanese))
OE-006 The beneficial effects of natriuretic peptides following myocardial infarction are mediated partly by suppression of the renin-angiotensin system(Acute Myocardial Infarction, Basic 1 (IHD) : OE1)(Oral Presentation (English))
Role of Renin-Anglotensin System in Cardiac Remodeling after Aortic Regurgitation in Mice
ERK5 Plays a Critical Role in Cardiotrophin-1-Induced Hypertrophy of Neonatal Rat Ventricular Myocytes
Evidence for Cardiac Myocyte Regeneration in Injured Myocardium by Bone Marrow Derived Cells
JAB/SOCS1 and CIS3/SOCS3 inhibit hypertrophic response to cardiotrophin-1 independent of STAT3 suppression in neonatal rat ventricular myocytes
Overxepression of brain natriuretic peptide mediates cardiac matrix metalloproteinase induction and induces cardiac rupture after acute myocardial infarction in mice
Inhibition of histone deacetylase selectively induces expression of cardiac embryonic genes that contain neuron-restrictive silencer element in cultured ventricular myocytes
Disruption of natriuretic peptides receptor A leads to higher mortality and exaggerated left ventricular remodeling after myocardial infarction in mice
Genetic disruption of angiotensin II type 2 (AT2) receptor worsened short-term survival after acute myocardial infarction in mice
Natriuretic peptides induce NF-κ B activation during myocardial ischemia-reperfusion : possible mechanisms of acute inflammation and coronary endothelial apoptotic cell death

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