Takano Hitoshi | the First Department of Internal Medicine, Nippon Medical School

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概要

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著作論文

Pharmacological Preconditioning With Bradykinin Affords Myocardial Protection Through NO-dependent Mechanisms
Increased Peripheral Blood Mononuclear Cells Is an Important Determinant of Left Ventricular Remodeling in Patients with Acute Myocardial Infarction
PE-476 Increased Peripheral Blood Mononuclear Cell Count is an Independent Predictor for Cardiac Events in Patients with Acute Myocardial Infarction(Acute myocardial infarction, clinical(diagnosis/treatment)(07)(IHD),Poster Session(English),The 72nd Annua
PE-069 Metabolic Syndrome Increases the Risk of Mortality in Patients with Acute Coronary Syndromes Underwent Successful Percutaneous Coronary Intervention(Diabetes/Obesity/Metabolic syndrome(04)(H),Poster Session(English),The 72nd Annual Scientific Meeti
OE-167 Impaired Coronary Microcirculation is a Predictor of Cardiac Events in Patients with Non-Ischemic Cardiomyopathy(Heart failure, clinical-1 (M) OE28,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulati
FRS-102 Pharmacological Modification on Left Ventricular Hemodynamics in Patients with Hypertrophic Obstructive Cardiomyopathy to Evaluate Cases for Percutaneous Septal Myocardial Ablation(Frontier of Cardiomyopathy Research-2 (M) FRS21,Featured Research
PJ-642 Current Composite Therapeutic Strategy for Chronic Cardiac Failure Due to Hypertrophic Obstructive Cardiomyopathy(Cardiomyopathy, basic/clinical-7 (M) PJ108,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Ci
OE-257 Peak White Blood Cell Count and Fasting Glucose are Independent Predictors of Left Ventricular Remodeling in Reperfused Acute Myocardial Infarction(Acute myocardial infarction, clinical (diagnosis/treatment)-1, The 71st Annual Scientific Meeting of
OE-254 Peak White Blood Cell Count and Fasting Glucose are Independent Predictors of Cardiac Events in Patients with Acute Myocardial Infarction(Acute myocardial infarction, clinical (pathophysiology)-1, The 71st Annual Scientific Meeting of the Japanese
OJ-357 Evaluation of functional and emotional status long after catheter or surgical coronary revasculization in Octogenarian with severe coronary artery disease(Coronary Revascularization, PTCA/Stent/DCA/Rotablator/New Device 9 (IHD) : OJ43)(Oral Present
Relation between Serum CK Release and Result of Created Myocardial Necrosis in Hypertrophic Obstructive Cardiomyopathy Undergoing Percutnaeous Myocardial Alcohol Ablation
OE-004 Metabolic Syndrome Increases Atherosclerotic Plaque Burden and Cardiac Events Associated with Inflammation in Patients with Acute Coronary Syndromes(Acute coronary syndrome, basic/clinical-1 (IHD) OE1,Oral Presentation (English),The 70th Anniversar
Temporary balloon occlusion halfly predicts reduction of pressure gradient in percutaneous catheter treatment with alcohol for hypertrophic obstructive cardiomyopathy (HOCM)
Increased coronary atherosclerotic plaque burden is an important determinant of adverse outcomes in diabetic patients after percutanous coronary intervention
OE-176 Statins therapy attenuates monocyte proinflammatory cytokines production by C-reactive protein and reduces cardiac events in patients with chronic heart failure(Heart Failure, Clinical 1 (M) : OE22)(Oral Presentation (English))
C-reactive protein synergizes with interferon-γ to upregulate monocyte tissue factor and factor VII
OJ-251 Improvement of Endothelial Dysfunction Indicated Early Phase Recovery after Bone Marrow Vascular Regenerative Therapy in Human(Vascular smooth muscles-1 (H) OJ42,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Jap
Effectiveness and safety of percutaneous catheter alcohol ablation for medically refractory patients with hypertrophic obstructive cardiomyopathy : single center experience
Ongoing Myocardial Damage in Patients with Chronic Heart Failure Is Strongly Related to Upregulated Monocyte Proinflammatory Cytokine Production
Upregulated Monocyte Proinflammatory Cytokine Production by C-Reactive Protein Contributes to the Exacerbation of Chronic Heart Failure

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