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When an artificial heart is pumping, the shutting of valves produces water hammers in which the peak value of the pressure may become unphysiologically high so as to injure the tissue directly. In this report, the propagation of this extraordinary pressure wave in arteries is investigated theoretically and experimentally in vitro and in vivo. The wave produced by the outlet valve shutting reaches a pressure two times higher than the systolic pressure near the valve part and the canulla of artificial heart. In the course of propagation into arteries, this wave attenuates and its pressure becomes lower than the systolic pressure. Hence, the extraordinary high pressure due to water hammer does not seem to damage the tissue directly. But the rate of attenuation of this wave is relatively small in the arteries, and the. amplitude does not decrease much more over the distance of 20 cm and 8 cm respectively away from the canulla of the artificial heart in descending aorta and in pulmonary artery. The influence of this surviving wave on the living body is still a remaining problem when an artificial heart is used for a long time. For the purpose of avoiding the high pressure due to water hammer, we obtained the guiding principle for pump design and physical properties of the materials for artificial heart.
一般社団法人日本生体医工学会の論文