Ataxic Hemiparesis Following Thalamic Lacunar Infarction.
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概要
- 論文の詳細を見る
A 60-year-old man developed left hemiparesis and homolateral ataxia with normal sensation and normal somatosensory evoked potentials. A lacunar infarct with gadolinium enhancement in the right dorsolateral part of the thalamus was demonstrated on magnetic resonance imaging. Thalamic lesion is a relatively rare cause of ataxic hemiparesis ; most of the reported cases of ataxic hemiparesis caused by thalamic lesion were accompanied by sensory disturbances. This is an interesting case which suggested that the thalamic lesion could be responsible for the ataxic hemiparesis without a sensory disturbance.(Internal Medicine 31 : 889-892, 1992)
- 社団法人 日本内科学会の論文
著者
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Sawada Tohru
The Cerebrovasular Division Department Of Medicine And The Department Of Radiology National Cardiova
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Imakita Satoshi
Department Of Radiology National Cardiovascular Center
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KURIYAMA Yoshihiro
the Cerebrovascular Division, Department of Medicine, National Cardiovascular Center
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EMORI Tetsuro
the Cerebrovascular Division, Department of Medicine, National Cardiovascular Center
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SAWADA Tohru
the Cerebrovascular Division, Department of Medicine, National Cardiovascular Center
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- Ataxic Hemiparesis Following Thalamic Lacunar Infarction.