失語並びにその近縁症状の剖検的所見

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Autopsy findings of the cases of aphasia and its related disease were correlated with clinical picture to study the mechanism of speech. A total of 9 patients seen in our clinic during the period from 1942 to 1958 constitute the basis of this report. (1) Sensory aphasia occurs due to a lesion in the nerve center in the central one-third of the area of sinistral gyrus temporalis superior. Some cases in which the disturbanec involved greater part of the gyrus angularis and gyrus supramarginalis were markedly free from the symptoms of aphasia. This seems to indicate that those two regions are not the center concerned in the development of aphasia. (2) An injury to the gyrus angularis causes agnosia, alexia and apraxia. A lesion in the capusula interna also causes apraxia, indicating it is also concerned in the developmen of apraxia. (3) Cerebral dominance becomes well established before a person comes of age. The time at which cerebral dominance is well established greatly varies from person to person depending on constitutional differences, environmental conditions and other factors. Excitement of the nerve center in the recessive hemisphere is weak, and when the dominant hemisphere suffers a severs injury, the expression by speech consists only parrot-like imitation of voice, words expressing sensation, and words in wrong and senseless combinations. This seems to represent an aspect of compensatoay function of the recessive hemisphere, and also to indicate that the dominant hemisphere usually regulates and controls the recessive hemisphere.
1960-09-28

失語並びにその近縁症状の剖検的所見

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