4 Signal Tranduction Pathways Towards Cell death in Cardiac Remodeling(Molocular Mechanism of Heart Failure,Meet the Expert 5 (ME5) (M),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)

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FRS-071 Constitutive Autophagy Is Required to Maintain Cardiac Structure and Function in Aged Mouse Heart(FRS15,Novel Molecular Mechanisms of Heart Failure 1 (M),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japanese Circul
FRS-061 Cardiac-specific Disruption of Calpain4 Revealed Its Cardioprotective Role in Response to Pressure Overload(FRS13,Novel Molecular Mechanisms of Heart Failure 2 (M),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japan
PJ-864 Signs of circulatory failure early hours after PCPS may predict prognosis in patients with CPAOA and PCPS(Emergency care / CPR(05)(H),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PJ-054 Increased Cardiac Glucocorticoid Contribute to Promoting Cardiac Hypertrophy(Cardiac hypertrophy, basic/clinical(02)(M),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PE-334 IKKβ/NF-κB Pathway Protects Hearts from Hemodynamic Stress Mediated through Regulating MnSOD Expression(Heart failure, basic(03)(M),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OE-255 Cross-talk between p38 Dependent Pathological Remodeling Cascade and Cardiac Adaptive Responses(Cardiac hypertrophy, basic/clinical(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
5 Role of Autophagy in Heart Failure(Novel Therapeutic Strategies against Heart Failure,KSC-JCS Joint Symposium (KSC-JCS Joint Symposium) (I),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
4 Signal Tranduction Pathways Towards Cell death in Cardiac Remodeling(Molocular Mechanism of Heart Failure,Meet the Expert 5 (ME5) (M),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Presenilin 2 Regurates Excitation-contraction Coupling in Hearts(Excitation-contraction Coupling/Ion Channel 1 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-524 The Protective Role of STAT6 in Response to Pressure Overload in Vivo(Molecular Biology, Myocardium 2 (M) : OJ64)(Oral Presentation (Japanese))
OJ-023 Cardiac-specific Over-expression of Sarcolipin Inhibits the Sarcoplasmic Reticulum Ca^ ATPase (SERCA2a) and Reduces the Cardiac Function in Mice(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
OJ-020 Presenilin 2 Directly Interacts with Cardiac Ryanodine Receptor(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
OE-367 The Ca^/calmodulin-dependent Protein Kinase II Induces Cardiac Myocyte Hypertrophy through the Activation of Apoptosis Signal-regulating Kinase 1 and Nuclear Factor-κB(Cardiac Hypertrophy, Basic and Clinical 1 (M) : OE46)(Oral Presentation (Eng
Beta-Adrenergic Stimulation Failed to Regulate Intracellular Ca^ Concentration and Isometric Tension in SERCA2a Overexpressing Heart
Cardiac-Specific Overexpression of a High Ca^ Affinity Mutant of Cardiac Ca^ ATPase Attenuates in Vivo Pressure Overload Cardiac Hypertrophy
The Small G Protein Rac1 Induce Cardiac Myocyte Hypertrophy through the Activation of Apoptosis Signal-Regulating Kinase1 and Nuclear Factor-Kappa B
Cardiac-Specific Disruption of Raf-1 Gene Induces Apoptosis and Cardiac Dysfunction
The Antioxidant N-2-Mercaptopropionyl Glycine Attenuates Left Ventricular Hypertrophy in Murine Pressure-Overloaded Model
Disruption of a single copy of the p38alpha mitogen-activated protein kinase gene leads to cardioprotection against ischemia-reperfision
Involvement of ASK1 in G-protein coupled receptor agonistinduced cardiac hypertrophy
Amino acids ^KDDKPV^ in the cytosolic domain of SERCA2a are critical for functional association with PLN in vivo
PE-433 Reduction in Hemoglobin-Oxygen Affinity Results in the Improvement of Exercise Capacity in Mice with Chronic Heart Failure(Heart failure, basic-4, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-046 ASK1-p38 MAP Kinase Pathway Plays an Important Role in Physiological Hypertrophy Induced by Exercise Training(Cardiac hypertrophy, basic/clinical-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-066 Autophagy in Cardiomyocyte is Essential for Maintenance of Cardiac Function(Heart Failure, Cardiac Remodeling and Cardiomyopathy 1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-068 Cardiac-specific Activation of Apoptosis Signal-regulating Kinase 1 Leads to Cardiac Dysfunction through both Apoptotic and Non-apoptotic Cell Death(Cell death, apoptosis/necrosis (M) OE12,Oral Presentation (English),The 70th Anniversary Annual Sci
OE-067 Non-apoptotic Cell Death in Cardiac Injury : The Role of Cyclophilin D(Cell death, apoptosis/necrosis (M) OE12,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
4 The Role of Mitogen-activated Protein Kinase Family in the transition to Heart Failure(AHA/JCS Joint Symposium (AHA) (M) : Molecular Mechanism of Heart Failure)(Special Program)
FRS-015 Genetic Modulation of Ca^ Uptake Sarcoplasmic Reticulum Does Not Affect Maximal Ca^ Content and Ca^ Leakage in Mouse Myocardium(Cardiac Function "Ventricular Mechanics and Calcium Handling", The 71st Annual Scientific Meeting of the Ja
OJ-296 Overexpression of SERCA2a and Sarcolipin Does Not Affect Maximal Ca^ Content and Ca^ Leakage of Sarcoplasmic Reticulum in Mouse Myocardium(Cardiac function, basic/clinical-2 (M) OJ50,Oral Presentation (Japanese),The 70th Anniversary Annual
OE-132 Gab Family Proteins Are Essential for the Prevention of Heart Failure(Heart failure, basic-1 (M) OE22,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
PJ-646 Usefullnes of "Off-pump Trial" and "Volume-loading Trial" : To Identify Who Could be Weaned from Left Ventricular Assist Device(PJ109,Heart Failure (Treatment) 3 (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circu
FRS-075 Chronic Inhibition of Apoptosis Signal-regulating Kinase 1 (ASK1) by Myocardial Gene Transfer Suppressed Progression of Heart Failure in Genetic Cardiomyopathy(Heart Failure Research (M) FRS15,Featured Research Session,The 70th Anniversary Annual
OE-330 Oxidative Stress Causes Heart Failure with Impaired Mitochondrial Respiration(Molecular biology, myocardium-1 (M) OE56,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Cell Death in Heart Failure(Molecular Mechanisms)
Involvement of Activation of ASK1 and NF-κB in the δ3 Isoform of Ca^/calmodulin-dependent Protein Kinase II Induced Cardiac Myocyte Hypertrophy(Cardiac Hypertrophy 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
p38α is Involved in the Mechanism of β-adrenergic Stress-induced Cardiomyocyte Cell Death and Cardiac Dysfunction(Molecular Biology, Myocardium 1 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Novel In Vivo Tool to Evaluate Autophagic Activity in the Heart
Cardiac Specific Disruption of P38α Gene Leads to Left Ventricular Dysfunction in Pressure Overload in Mice
OE-077 Downregulation of Ferritin Heavy Chain is an Important Trigger of Cardiac Cell Death and Progression of Heart Failure(Heart failure, basic(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
1 Non-apoptotic Cardiomyocyte Death as a Cause of Cardiac Remodeling(Cardiomyocyte Death and Cardiac Remodeling,Symposium 9 (SY-09) (M),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
The Antioxidant Edaravone Attenuates Pressure Overload-Induced Left Ventricular Remodeling (Cardiac Hypertrophy, Basic (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)

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