心畸形三例(牛)

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Case 1 (Fig. 1, Schema 1) The heart was obtained from 2 year old Holstein-Friesian-hydrid cow, bled to death. After slight body movement it used to tire easily and squat or fall down to rest on the ground, showing severe dyspnoe and cyanosis. After a short recess, it recovered and could walk as usual. Any other clinical observations are unknown. The pathologico-anatomical diagnosis of the heart follows; Peristent truncus arteriosus communis, partial type (Humphreys); Basal defect of ventricular septum with some small holes in its muscular portion; Patent foramen ovale (small slit); High grade compensatory hypertrophy of the right ventricle. Case 2 (Fig. 2, 3, Schema 2, 4) A29 month old Japanese Black Breed cow. Body length 1.1m., Height 1.1m., Weight 165kg. Middle sized stature. Nutrition was not so bad. It staggered at walking and seemed slightly atactic. Conjunctiva was more or less cyanotic. Hair appeared rather dull and ruffled. It was easily excitable even by slight stimuli, the heart having been accelerated and palpable with fingers. Heartsounds were increased in intensity and intracardial murmurs were noted. Frequent but weak pulsation counted 66p.m. The jugnlar vein pulsation was remarkable. The respiartion was also accelerated by stimuli, resulting labored respiration or dyspnoe. The blood was dark red in color, Hb 43%, Red corpuscles 4,580,000, Leucocytes 5,200. The urine was straw-yellow, clear, spec. grav. 1,022, Albumin (±), Sugar (-), Bilirubin (-), Hb (-). Clinical investigation shows easy excitability with accelerated respiration even when any one draws near. It dislikes physical movement, and after being forced into light walking it gives rise to dyspnoe, markedly frequent pulsation which results in dropping on the knee with dizziness, severe dyspnoe, jugular vein pulsation and marked acceleration of heart beats. The pathologico-anatomical diagnosis of the heart follows: Basal defect of the ventricular septum; Dextroposition of the dilated and hypertrophic aorta ("Rider" position); Sinistropositon of the hypoplastic pulmonary artery; Patent ductus Botalli (Adossement ALMAGROS or Type b in the classification after Vierordt); Patent foramen ovale; Partial abnormal course of heart artery and vein; Compensatory hypertrophy of right ventricle. Case 3 (Fig. 4, 5, 6, Schema 3) A Holstein-Friesian breed, new-born female calf. Smaller stature than mormal. It tired easily with severe palpitaion. Intracardial murmur was heard. Valvular disease was suspected. It died on the fifth day after parturion. The pathologico-anatominal diagnosis of the heart follows; Abnormal course of pulmonary vein (no connection with the left atrium, but presumably with the right through coronary sinus in unity with the vena hemiazygos); Hypoplasia of the left heart system (left atrium, ventricle and aorta); Compensatory hypertrophy of the right heart system (right atrium, ventricle and pulmonary artery); Patent foramen ovale; Patent ductus Botalli (type a in the classification after Vierordt).
1955-12-25

心畸形三例(牛)

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