Ultrastructural localization of hyarulonic acid in the otolithic organ of the mongolian gebril as revealed by enzyme-gold technique
スポンサーリンク
概要
著者
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Nakae Susumu
Department Of Pathology Stanford University School Of Medicine
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Nakae Susumu
Department Of Otplaryngology Matsushita Memormal Hospital
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Nakae Susumu
Department Of Allergy And Immunology National Research Institute For Child Health And Development
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Tachibana Masatoshi
Department of Otplaryngology, Meiji College of Oriental Medicine
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Tachibana Masatoshi
Department Of Otplaryngology Meiji College Of Oriental Medicine
関連論文
- Th17 and Allergy
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- IL-1β, but not IL-1α, is required for antigen-specific T cell activation and the induction of local inflammation in the delayed-type hypersensitivity responses
- Abnormal T cell activation caused by the imbalance of the IL-1/IL-1R antagonist system is responsible for the development of experimental autoimmune encephalomyelitis
- IL-1 is required for allergen-specific T_h2 cell activation and the development of airway hypersensitivity response
- IL-1-induced tumor necrosis factor-α elicits inflammatory cell infiltration in the skin by inducing IFN-γ-inducible protein 10 in the elicitation phase of the contact hypersensitivity response
- Ultrastructural localization of hyarulonic acid in the otolithic organ of the mongolian gebril as revealed by enzyme-gold technique
- Amphiregulin is Not Essential for Induction of Contact Hypersensitivity
- Amphiregulin Is Not Essential for Ovalbumin-Induced Acute Airway Inflammation in Mice
- Development of IL-17-mediated Delayed-Type Hypersensitivity Is Not Affected by Down-Regulation of IL-25 Expression
- IL-33 and IL-33 Receptors in Host Defense and Diseases