Presence of Endothelial Dysfunction Is the Major Determinant of Maladaptive Vascular Remodeling in Brachial Arteries of Untreated Hypertensive Patients
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概要
- 論文の詳細を見る
- 社団法人日本循環器学会の論文
- 2003-03-01
著者
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Kitabatake Akira
Department of Cardiovascular Medicine, Graduate School of Medicine, Hokkaido University
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Onozuka Hisao
Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine
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FURUMOTO Tomoo
Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine
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GOTO Kazutomo
Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine
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YAMADA Satoshi
Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine
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Gotoh Kazutomo
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Wu Di
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Onozuka Hisao
Faculty Of Health Sciences Hokkaido University Graduate School
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FUJII Satoshi
Department of Neurosurgery, Yokohama City University School of Medicine
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Goto Kazutomo
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Yamada Satoshi
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Furumoto Tomoo
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Komuro Kaoru
Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine
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Mikami Taisei
Faculty Of Health Sciences Hokkaido University Graduate School
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Mikami Taisei
College Of Medical Technology Hokkaido University
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Nishihara Keiko
Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine
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Sobel E
Department of Medicine, University of Vermont
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Kitabatake Akira
Department Of Cardicvascular Medicine Hokkaidou University
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Fujii Satoshi
Department Of Applied Chemistry Faculty Of Engineering Himeji Institute Of Technology
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Nishihara Keiko
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Sobel E
Department Of Medicine University Of Vermont
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Komuro Kaoru
Department Of Clinical Research Hakodate National Hospital
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Komuro Kaoru
Department Of Cardiovascular Medicine Date Red Cross Hospital
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Onozuka Hisao
Department Of Cardiovascular Medicine Hokkaido University Hospital
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Onozuka Hisao
Department Of Cardiovascular Medicine Graduate School Of Medicine Hokkaido University
関連論文
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- Osteopontin is Essential for Angiotensin II-Induced Cardiac Fibrosis(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- A Newly Developed Angiotensin II Type 1 Receptor Antagonist, CS866, Promotes Regression of Cardiac Hypertrophy by Reducing Integrin β_1 Expression
- Angiogenesis Is Altered by Angiotensin Type 1 Receptor Blockade in Cardiomyopathic Hamster Hearts
- Angiotensin II Type 2 Receptor Expression Affects Remodeling after Myocardial Infarction in Mice
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Blockade of T-cell costimulatory signals using adenovirus vectors attenuates experimental autoimmune myocarditis through the induction of antigen-specific T-cell anergy
- The Chronic Effect of Amlodipine on Cardiac Remodeling in Cardiomyopathic Hamster Hearts
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- Development of Atherosclerosis in Osteopontin Transgenic Mice
- Immunosuppressive Therapy in Refractory Heart Failure : Blockade of T Cell Costimulatory Signals
- Role of Microvasculature in Cardiac Remodeling Process
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
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- Prevalence and Clinical Characteristics of Patients With Atrial Fibrillation : Analysis of 20,000 Cases in Japan
- The Efficacy of Radiofrequency Catheter Ablation for the Treatment of Ventricular Tachycardia Associated With Cardiomyopathy
- A Case of Adenosine-Sensitive Ventricular Tachycardia in a Very Elderly Male
- A Case of Familial Ventricular Tachycardia
- Norepinephrine Disrupts Cytoskeletal Framework of Microtubules in Rat Hearts : COMPENSATORY MECHANISMS AND THEIR LIMITATIONS IN HEART FAILURE
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- Experimental Study of the Relationship between Perfluoro-Octyl Bromide Emulsion and Norepinephrine Release in Reperfusion Arrhythmia : Isolated Guinea Pig Heart Model
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