Hypofibrinolysis and Increased Metalloproteinase Expression Associated with Coronary Vasculopathy in NIDDM : Primary Role of Angiotensin II Type 1 Receptor
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概要
- 論文の詳細を見る
- 社団法人日本循環器学会の論文
- 2003-03-01
著者
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Kitabatake Akira
Department of Cardiovascular Medicine, Graduate School of Medicine, Hokkaido University
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Hattori Yuichi
Department of Cardiovascular Medicine, Hokkaido University graduate School of Medicine
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Sakuma Ichiro
北海道大学 医学研究科循環病態内科学分野
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Sakuma Ichiro
Department Of Cardiovascular Medicine Hokkaido University School Of Medicine
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Sakuma Ichiro
Dept. Of Pharmacology Hokkaido University Graduate School Of Medicine
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Subrina Jesmin
Division Of Gene Therapeutics Research Institute International Medical Center Of Japan
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Subrina Jesmin
Center For Tsukuba Advanced Research Alliance (tara) University Of Tsukuba
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Jesmin Subrina
Department Of Gene Diagnostics And Therapeutics Research Institute International Medical Center Of J
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Jesmin Subrina
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Hattori Yuichi
Dept. Of Pharmacology Hokkaido University Graduate School Of Medicine
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Hattori Yuichi
Department Of Applied Mathematics Faculty Of Science Konan University
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Kitabatake Akira
Department Of Cardicvascular Medicine Hokkaidou University
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Sakuma Ichiro
Department Of Cardiovascular Medicine Graduate School Of Medicine Hokkaido University
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Sakuma Ichiro
Department Of Cardiovascular Medicine School Of Medicine Hokkaido University
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Jesmin Subrina
Center For Tsukuba Advanced Research Alliance (tara) University Of Tsukuba:institute Of Clinical Med
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Hattori Yuichi
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Sakuma Ichiro
Univ. Tokyo
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Jesmin Subrina
Department of Cardiology, National Center for Global Health and Medicine
関連論文
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- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
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- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
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