PIP_3-Inhibition of RGS-Action and Its Reverse by Ca^<2+>/Calmodulin Mediate Voltage-Dependent Control of G Protein-Cycle to a Cardiac K^+ Channel
スポンサーリンク
概要
- 論文の詳細を見る
- 社団法人日本循環器学会の論文
- 2003-03-01
著者
-
Ishii Masaru
Department Of Pharmacology Ii Graduate School Of Medicine Osaka University
-
Karachi Yoshihisa
Department Of Pharmacology Osaka University Graduate School Of Medicine
関連論文
- Superconducting State of Silver-Oxide Clathrate Ag_6O_8AgNO_3(Condensed matter: electronic structure and electric, magnetic, and optical properties)
- Possibility of Preventive Treatment for EBV-associated NK Cell-lineage Proliferative Disorders
- Intracellular Ca^-elevation Plays a Crucial Role in the PKC-independent Antagonizing Effect of Angiotensinll against Insulin-induced Production of PI(3, 4, 5)P3 in Cardiomyocytes (Molecular Biology, Myocardium 2 (M), The 69th Annual Scientific Meeting
- A case of Mikulicz's disease with Th2-biased cytokine profile : possible feature discriminable from Sjogren's syndrome
- Expression and Function of Transmembrane-4 Superfamily (Tetraspanin) Proteins in Osteoclasts : Reciprocal Roles of Tspan-5 and NET-6 during Osteoclastogenesis
- Ca^ Elevation Evoked by Membrane Depolarization Regulates G Protein Cycle via RGS Proteins in the Heart
- Ca^ -elevation evoked by membrane-depolarization regulates G protein-cycle via RGS proteins in the heart
- Osteoclast cell fusion : mechanisms and molecules
- A Rapid Voltage and Time-Dependent Quinidine-Block of HERG Channels Causes Reverse Frequency-Dependency
- PIP_3-Inhibition of RGS-Action and Its Reverse by Ca^/Calmodulin Mediate Voltage-Dependent Control of G Protein-Cycle to a Cardiac K^+ Channel
- PE-010 Allosteric Modulation of RGS Proteins by a Phospholipid and Ca^/Calmodulin in Native Cardiac Myocytes(Arrhythmia, Basic 3 (A) : PE2)(Poster Session (English))