IS113 MYOCARDIAL ISCHEMIA UPREGULATES Kir6.2 mRNA, A PUTATIVE CARDIAC K_<ATP> CHANNEL GENE
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概要
- 論文の詳細を見る
- 社団法人日本循環器学会の論文
- 1997-03-05
著者
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Nawada R.
Cardiovascular Division Of Internal Medicine Faculty Of Medicine Kyoto University
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TAKANO M.
Department of Orthopaedic Surgery, Sapporo Medical University
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Horie M.
Department Of Cardiovascular Medicine Kyoto University Graduate School Of Medicine
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Kouchi I.
Cardiovascular Division Of Internal Medicine Faculty Of Medicine Kyoto University
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Akao M.
Cardiovascular Division of Internal Medicine, Faculty of Medicine, Kyoto University
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Otani H.
Cardiovascular Division of Internal Medicine, Faculty of Medicine, Kyoto University
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Murakami T.
Cardiovascular Division of Internal Medicine, Faculty of Medicine, Kyoto University
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Horie M.
Cardiovascular Division of Internal Medicine, Faculty of Medicine, Kyoto University
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Morita Y.
Cardiovascular Division of Internal Medicine, Faculty of Medicine, Kyoto University
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Sasayama S.
Cardiovascular Division of Internal Medicine, Faculty of Medicine, Kyoto University
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Sasayama S.
Department Of Cardiovascular Medicine Kyoto University Graduate School Of Medicine
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Akao M.
Cardiovascular Division Of Internal Medicine Faculty Of Medicine Kyoto University
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Otani H.
Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine
関連論文
- P-25 Free Fat Graft And Corrective Osteotomy for the Delta Phalanx.
- IS113 MYOCARDIAL ISCHEMIA UPREGULATES Kir6.2 mRNA, A PUTATIVE CARDIAC K_ CHANNEL GENE
- IS120 BASAL ACTIVITY OF TYROSINE KINASE KEEP DELAYED-RECTIFIER K CHANNELS OPEN IN GUINEA PIG VENTRICULAR CELLS.
- IS111 CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR (CFTR) CONFERS THE SENSITIVITY TO SULFONYLUREAS ON KIR6.1, AN INWARDLY-RECTIFYING K CHANNEL, EXPRESSED IN NIH3T3 CELLS.
- IS112 PROPERTIES OF RECONSTITUTED ATP-SENSITIVE POTASSIUM CHANNELS