Effects of Angiotensin Converting Enzyme Inhibitor and β-Blocker on Autonomic Nervous System Activity in Idiopathic Dilated Cardiomyopathy
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概要
- 論文の詳細を見る
- 社団法人日本循環器学会の論文
- 1994-06-20
著者
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Kitabatake Akira
Department of Cardiovascular Medicine, Graduate School of Medicine, Hokkaido University
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Kohya Tetsuro
NTT East Japan Sapporo Hospital
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Kohya Tetsuro
Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine
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Tomita Fumishi
Department of Cardiovascular Medicine, Hokkaido University, Graduate School of Medicine
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Kaji Tohru
Department of Cardiovascular Medicine, Hokkaido University, Graduate School of Medicine
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ONO Tomohide
Department of Cardiovascular Medicine, Hokkaido University, School of Medicine
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Ono Tomohide
Departments Of Cardiovascular Medicine Hokkaido University School Of Medicine
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Kitabatake Akira
Department Of Cardicvascular Medicine Hokkaidou University
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Kohya Tetsuro
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Kaji Tohru
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Tomita Fumishi
Department Of Cardiovascular Medicine Hokkaido University Graduate School Of Medicine
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Kitabatake Akira
Departments Of Cardiovascular Medicine Hokkaido University School Of Medicine
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Tsuzuki Noriko
Department of Cardiovascular Medicine, Hokkaido University School of Medicine
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Tsuzuki Noriko
Department Of Cardiovascular Medicine Hokkaido University School Of Medicine
関連論文
- 1 Pharmacogenomics of β-blocker Therapy in Chronic Heart Failure(New Approaches to Heart Failure-from Pharmacogenomics to Drug Development, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- Which is most Beneficial for Preserving Cardiac Function and Coronary Microvasculaturein DCM, ACE-I, ARB or Two in Combination? (Cardiac Hypertrophy/Cardiomyopathy 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Eplerenone Prevents Cardiac Fibrosis through the Inhibition of OPN Expression (Cardiac Hypertrophy, Basic (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Osteopontin is Essential for Angiotensin II-Induced Cardiac Fibrosis(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- A Newly Developed Angiotensin II Type 1 Receptor Antagonist, CS866, Promotes Regression of Cardiac Hypertrophy by Reducing Integrin β_1 Expression
- Angiogenesis Is Altered by Angiotensin Type 1 Receptor Blockade in Cardiomyopathic Hamster Hearts
- Angiotensin II Type 2 Receptor Expression Affects Remodeling after Myocardial Infarction in Mice
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Blockade of T-cell costimulatory signals using adenovirus vectors attenuates experimental autoimmune myocarditis through the induction of antigen-specific T-cell anergy
- The Chronic Effect of Amlodipine on Cardiac Remodeling in Cardiomyopathic Hamster Hearts
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Coronary microvasculature is related with cardiac remodeling in cardiomyopathic hearts
- Development of Atherosclerosis in Osteopontin Transgenic Mice
- Immunosuppressive Therapy in Refractory Heart Failure : Blockade of T Cell Costimulatory Signals
- Role of Microvasculature in Cardiac Remodeling Process
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Complete Freund's Adjuvant Can Activate TLR2 and Suppress Cardiac Necrosis in Cardiomyopathic Heart
- Complete Freund's Adjuvant can Suppress Cardiac Remodeling in Cardiomyopathic Heart, and Induce Pulmonary Granulomas
- Prognosis and Prognostic Factors in Patients With Idiopathic Dilated Cardiomyopathy in Japan : Results From a Nationwide Study
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- Exercise Training and Weight Reduction Decreased C-Reactive Protein Levels in Overweight Women
- Cross-sectional study of exercise habit and cardiovascular risk factors in a general population
- Differential response of skeletal muscle sympathetic nerve activity and heart rate to alteration in arterial oxygenation
- The relationship between lactate-transport and Monocarboxylate transporter1 (MCT1) in red blood cell in Normal subjects (NL) and patients with CHF
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- Increased cycle length variability during ventricular fibrillation : a novel predictor of arrhythmia recurrence
- Altered repolarization dynamics during premature stimulation in Brugada syndrome with inducible ventricular fibrillation
- Prevalence and Clinical Characteristics of Patients With Atrial Fibrillation : Analysis of 20,000 Cases in Japan
- The Efficacy of Radiofrequency Catheter Ablation for the Treatment of Ventricular Tachycardia Associated With Cardiomyopathy
- A Case of Adenosine-Sensitive Ventricular Tachycardia in a Very Elderly Male
- A Case of Familial Ventricular Tachycardia
- Norepinephrine Disrupts Cytoskeletal Framework of Microtubules in Rat Hearts : COMPENSATORY MECHANISMS AND THEIR LIMITATIONS IN HEART FAILURE
- OE-315 Salutary Effects of Attenuation of Angiotensin II Mediated JNK Signaling on Coronary Perivascular Fibrosis Associated With Insulin Resistance and Obesity(Diabetes/Obesity 2 (H) : OE40)(Oral Presentation (English))
- Natural Killer T Cells Accelerate Atherogenesis in Mice
- Up-Regulation of Plasminogen Activator Inhibitor-1 in HepG2 Cells by Interleukin (IL)-1 β and IL-6 and Its Attenuation by HMGCoA Reductase Inhibitor
- Up-Regulation of Plasminogen Activator Ihibitor-1 Production by Hepatocyte Growth Factar in Liver Cells and Its Potential Clinical Implications for Atherothrombosis
- Angiotensin Converting Enzyme (ACE) Inhibition Attenuates Hypofibrinolysis and Reduces Cardiac Perivascular Fibrosis during Development of Diabetic Cardiomyopathy in Obese Mice
- Induction of Plasminogen Activator Inhibitor-1 in Endothelial Cells by Basic Fibroblast Growth Factor and its Modulation by Fibric Acid
- Hypofibrinolysis Downregulates Coronary Capillary Network Remodeling in Aged Obese Diabetic Rats : Implications for Increased Myocardial Vulnerability to Ischemia in Diabetes
- Proinflammatory Cytokines, lnterleukin-1 and -6, and HMGCoA Reductase Inhibitor Up-regulate Plasminogen Activator Inhibitor-1 Production in a Human Liver Cell Line
- Induction of Tissue Factor in Human Endothelial Cells by Basic Fibroblast Growth Factor and its Modulation by Fibric Acid
- Novel Polyethylene Glycol Conjugated S-Nitrosylated Hemoglobin Potently Inhibits Platelet Activation and Adhesion to Artificial Surfaces : Therapeutic Implications for Platelet-Rich Thrombus
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- Reduced scavenger receptor activity in macrophages from atherosclerosis-resistant strain of mice
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- RT-PCR ANALYSIS OF TYPEL ANGIOTENSIN-II RECEPTOR WITH RELATION TO THE HUMAN HEART
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- The Role of Osteopontin in the Development of Granulomatous Lesions in Lung
- Angiotensin-converting enzyme gene polymorphism in Japanese patients with hypertrophic cardiomyopathy.
- Single-step isolation method for six glycoforms of human α_1-acid glycoprotein by hydroxylapatite chromatography and study of their binding capacities for disopyramide
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- Differential distribution of angiotensin II receptors in coronary microvasculature in diabetic heart : the potential clinical significance
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- Life-Style Related Factors and Idiopathic Dilated Cardiomyopathy : A Case-Control Study Using Pooled Controls
- Usefulness of Left Parasternal Approach for the Continuous-Wave Doppler Measurement of Left Ventricular Outflow Tract Pressure Gradient in Patients With Hypertrophic Cardiomyopathy
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- PJ-135 Feasibility of the Assessment of Left Ventricular Diastolic Function Using Doppler-Derived Early Diastolic Peak Strain Rate(Echo/Doppler 6 (I) : PJ23)(Poster Session (Japanese))
- PJ-020 Evaluation of Circumferential and Longitudinal Myocardial Contraction Using Tissue Doppler Strain Measurement in Patients with Generalized Left Ventricular Myocardial Impairment(Echo/Doppler 2 (I) : PJ4)(Poster Session (Japanese))
- Measuring Medium-Sized Muscular Arteries Using a Novel Broadband 15-MHz Linear Array Probe
- Usefulness of Color Tissue Doppler Imaging in the Assessment of Left Ventricular Diastolic Function : Comparison with Color M-Mode Doppler Technique
- Abnormal Diastolic Flow Velocity Pattern in the Distal Left Anterior Descending Coronary Artery in Patients with Apical Hypertrophy
- Experimental Study of the Relationship between Perfluoro-Octyl Bromide Emulsion and Norepinephrine Release in Reperfusion Arrhythmia : Isolated Guinea Pig Heart Model
- Induction of c-fos mRNA Expression by Pure Pressure Overload in Cultured Cardiac Myocytes
- Angiotensin converting enzyme inhibitors attenuated the expression of G-protein coupled receptor kinases in heart failure patients
- Chronic beta-adrenergic receptor stimulation enhances the expression of G-Protein coupled receptor kinases, GRK2 and GRK5, in both the heart and peripheral lymphocytes
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- CCAAT/enhancer Binding Protein δ is Critical for Induction of Plasminogen Activator Inhibitor-1 by Interleukin (IL)-1β and IL-6 in Hepatocytes(Infection/Inflammation/Immunity 2 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
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