Apoptosis of Human Tumor Cells by Chemotherapeutic Anthracyclines is Enhanced by Bax Overexpression
スポンサーリンク
概要
著者
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Yagi Takashi
Dept. Radiat. Genet. Grad. Sch. Med. Kyoto Univ.
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Yagi Takashi
Dept. Exp. Radiol. Fac. Medicine Kyoto Univ.
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LU anjun
Dept. Radiat. Genet., Grad. Sch. Med., Kyoto Univ.
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Lu Anjun
Dept. Radiat. Genet. Grad. Sch. Med. Kyoto Univ.
関連論文
- Types and Spectrum of the Mutations that Abolish the Activity of Cell Growth Inhibition in UV-Irradiated p21(sdi1/waf1/cip1) cDNA
- Expression of XPF/ERCC4 and ERCC1 in Normal Human and Group F Xeroderma Pigmentosum Cells.
- A protein deficient in xeroderma pigmentosum complementation group F cells.
- Construction of LacSwitch-Inducible p21 (sdi1/waf1/cip1) Expressing Clones.
- Inhibition of X-ray or Adriamycin-induced Apoptosis due to p21-Caused Blockage of G2/M Phase Transition
- Inhibition of X-ray or butyrolactone I-induced apoptosis by a CDK-specific inhibitor butyrolactone I.
- Mutation Specificity and Cancer
- A splicing mutation in exon 5 in the XPA gene
- A novel splicing mutation in the XPAC gene in a Japanese XP-A patient.
- Apoptosis of Human Tumor Cells by Chemotherapeutic Anthracyclines is Enhanced by Bax Overexpression
- DNA damages and mutations in UV irradlated normal Haras sequences.