AIM-1 induces chromosomal instability phenotype in p53-defective rodent cells
スポンサーリンク
概要
著者
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Han Zhen-bo
Res. Inst. Radiat. Biol. Med. Hiroshima Univ.
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SUZUKI Fumio
Res. Inst. Rad. Biol. Med. , Hiroshima Univ.
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TATSUKA Masaaki
Res. Inst. Radiat. Biol. Med. Hiroshima Univ.
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SUTO Shiho
Res. Inst. Radiat. Biol. Med. Hiroshima Univ.
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Suto Shiho
Molec. Radiobiol. Res. Inst. Radiat. Biol. Med. Hiroshima Univ.
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Suzuki Fumio
Molec. Radiobiol. Res. Inst. Radiat. Biol. Med. Hiroshima Univ.
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TATSUKA Masaaki
Molec. Radiobiol. Res. Inst. Radiat. Biol. Med. Hiroshima Univ.
関連論文
- Relative Biological Effectiveness of Accelerated Heavy Particles for Induction of In Vitro Transformation of Hamster Embryonic Cells.
- Involvement of AIM-1 in ionizing radiation-induced G2 checkpoint
- Molecular cloning of human MOB1, a factor involving in the mitotic spindle checkpoint pathway.
- AIM-1 induces chromosomal instability phenotype in p53-defective rodent cells
- Effects of Scid Mutation on Radiation Carcinogenesis - Modification of Radiation Carcinogenesis by A Genetic Factor -
- 149 Repression signal pathway of AIM-1/Aurora-B kinase activity distinct from ATM- and p53-mediated pathways is induced by ionizing radiation(Signal transduction, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- Probing the Dynamics of AIM-1/Aurora-B kinase in X-irradiated Living Cells during Mitosis and Cytokinesis