Alteration of Smad3 signaling in ischemic rabbit dermal ulcer wounds
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概要
- 論文の詳細を見る
- 2007-05-01
著者
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Schultz Gregory
Department Of Obstetrics And Gynaecology University Of Florida
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Mustoe Thomas
Division Of Plastic And Reconstructive Surgery Department Of Surgery Feinberg School Of Medicine Nor
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MUSTOE Thomas
Division of Plastic and Reconstructive Surgery, Wound Healing Research Laboratory, Feinberg School of Medicine, Northwestern University
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KLOETERS Oliver
Division of Plastic and Reconstructive Surgery, Wound Healing Research Laboratory, Feinberg School of Medicine, Northwestern University
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JIA Sheng-Xian
Division of Plastic and Reconstructive Surgery, Wound Healing Research Laboratory, Feinberg School of Medicine, Northwestern University
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ROY Nakshatra
Division of Plastic and Reconstructive Surgery, Wound Healing Research Laboratory, Feinberg School of Medicine, Northwestern University
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LEINFELLNER Gabrielle
Ludwig Boltzmann Institute, Research Center for Traumatology
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- Age-dependent response of primary human dermal fibroblasts to oxidative stress : cell survival, pro-survival kinases, and entrance into cellular senescence
- Symposium on Scarring: Papers from the Montpellier, France Meeting, May 2001
- Alteration of Smad3 signaling in ischemic rabbit dermal ulcer wounds
- Dynamic reciprocity in the wound microenvironment
- Antisense inhibition of connective tissue growth factor (CTGF/CCN2) mRNA limits hypertrophic scarring without affecting wound healing in vivo