Administration of IL-23 engages innate and adaptive immune mechanisms during fungal infection
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概要
- 論文の詳細を見る
- 2010-02-01
著者
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Straubinger Reinhard
Institute Of Immunology College Of Veterinary Medicine University Of Leipzig
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SCHUTZE Nicole
Institute of Immunology, College of Veterinary Medicine
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Sabat Robert
Interdisciplinary Group Of Molecular Immunopathology Dermatology/medical Immunology University Hospi
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Schuetze Nicole
Institute Of Immunology College Of Veterinary Medicine University Of Leipzig
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Alber Gottfried
Institute Of Immunology College Of Veterinary Medicine University Of Leipzig
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KLEINSCHEK Melanie
Institute of Immunology, College of Veterinary Medicine, University of Leipzig
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MULLER Uwe
Institute of Immunology, College of Veterinary Medicine, University of Leipzig
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STRAUBINGR Reinhard
Institute of Immunology, College of Veterinary Medicine, University of Leipzig
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BLUMENSCHEIN Wendy
Department of Experimental Pathology and Pharmacology, Schering-Plough Biopharma
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MCCLANAHAN Terrill
Department of Experimental Pathology and Pharmacology, Schering-Plough Biopharma
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KASTELEIN Robert
Department of Immunology, Schering-Plough Biopharma
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Muller Uwe
Institute Of Immunology College Of Veterinary Medicine University Of Leipzig
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Blumenschein Wendy
Department Of Experimental Pathology And Pharmacology Schering-plough Biopharma
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Mcclanahan Terrill
Department Of Experimental Pathology And Pharmacology Schering-plough Biopharma
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Kastelein Robert
Department Of Immunology Schering-plough Biopharma
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Kleinschek Melanie
Institute Of Immunology College Of Veterinary Medicine University Of Leipzig
関連論文
- Differential IL-23 requirement for IL-22 and IL-17A production during innate immunity against Salmonella enterica serovar Enteritidis
- IL-17A is produced by T_h17, γδ T cells and other CD4^- lymphocytes during infection with Salmonella enterica serovar Enteritidis and has a mild effect in bacterial clearance
- Administration of IL-23 engages innate and adaptive immune mechanisms during fungal infection
- IL-12 family members : differential kinetics of their TLR4-mediated induction by Salmonella Enteritidis and the impact of IL-10 in bone marrow-derived macrophages