Aberrant methylation of breast and ovarian cancer susceptibility gene 1 in chemosensitive human ovarian cancer cells does not involve the phosphatidylinositol 3'-kinase-Akt pathway
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概要
- 論文の詳細を見る
- 2010-07-10
著者
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WANG Yan-Qiu
Department of Obstetrics and Gynecology, Shanghai First People's Hospital, Shanghai Jiao Tong Univer
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ZHANG Jia-Rong
Department of Obstetrics and Gynecology, Shanghai First People's Hospital, Shanghai Jiao Tong Univer
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LI Shuang-Di
Department of Obstetrics and Gynecology, Shanghai First People's Hospital, Shanghai Jiao Tong Univer
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HE Yin-Yan
Department of Obstetrics and Gynecology, Shanghai First People's Hospital, Shanghai Jiao Tong Univer
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YANG Yi-Xia
Department of Obstetrics and Gynecology, Shanghai First People's Hospital, Shanghai Jiao Tong Univer
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LIU Xue-Lian
Department of Obstetrics and Gynecology, Shanghai First People's Hospital, Shanghai Jiao Tong Univer
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WAN Xiao-Ping
Department of Obstetrics and Gynecology, Shanghai First People's Hospital, Shanghai Jiao Tong Univer
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Wang Yan-qiu
Department Of Obstetrics And Gynecology Shanghai First People's Hospital Shanghai Jiao Tong Uni
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Wan Xiao-ping
Department Of Obstetrics And Gynecology Shanghai First People's Hospital Shanghai Jiao Tong Uni
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He Yin-yan
Department Of Obstetrics And Gynecology Shanghai First People's Hospital Shanghai Jiao Tong Uni
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Yang Yi-xia
Department Of Obstetrics And Gynecology Shanghai First People's Hospital Shanghai Jiao Tong Uni
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Liu Xue-lian
Department Of Obstetrics And Gynecology Shanghai First People's Hospital Shanghai Jiao Tong Uni
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Li Shuang-di
Department Of Obstetrics And Gynecology Shanghai First People's Hospital Shanghai Jiao Tong Uni
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Zhang Jia-rong
Department Of Obstetrics And Gynecology Shanghai First People's Hospital Shanghai Jiao Tong Uni
関連論文
- Aberrant methylation of breast and ovarian cancer susceptibility gene 1 in chemosensitive human ovarian cancer cells does not involve the phosphatidylinositol 3'-kinase-Akt pathway
- Estrogenic G protein-coupled receptor 30 signaling is involved in regulation of endometrial carcinoma by promoting proliferation, invasion potential, and interleukin-6 secretion via the MEK/ERK mitogen-activated protein kinase pathway