Late Na^+ current produced by human cardiac Na^+ channel isoform Na_v1.5 is modulated by its β_1 subunit
スポンサーリンク
概要
- 論文の詳細を見る
- Physiological Society of Japanの論文
- 2009-05-01
著者
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Maltsev Victor
Department Of Internal Medicine Cardiovascular Research Henry Ford Hospital Detroit
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KYLE John
Department of Neurobiology, Pharmacology, and Physiology, University of Chicago
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UNDROVINAS Albertas
Department of Internal Medicine, Cardiovascular Research, Henry Ford Hospital Detroit
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Kyle John
Department Of Neurobiology Pharmacology And Physiology University Of Chicago
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Undrovinas Albertas
Henry Ford Hospital Mi Usa
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Undrovinas Albertas
Department Of Internal Medicine Henry Ford Hospital
関連論文
- Late Na^+ current produced by human cardiac Na^+ channel isoform Na_v1.5 is modulated by its β_1 subunit
- Late sodium current contributes to diastolic cell Ca^ accumulation in chronic heart failure