Type 1 autoimmune hepatitis and adipokines : new markers for activity and disease progression?
スポンサーリンク
概要
- 論文の詳細を見る
- 2009-05-01
著者
-
Gambino Roberto
Department Of Internal Medicine University Of Turin
-
FLOREANI Annarosa
Department of Surgical and Gastroenterological Sciences, University of Padova
-
RIZZOTTO Erik
Department of Surgical and Gastroenterological Sciences, University of Padova
-
Rizzotto Erik
Department Of Surgical And Gastroenterological Sciences Gastroenterology University Of Padua
-
Floreani Annarosa
Department Of Surgical And Gastroenterological Sciences Gastroenterology University Of Padua
-
DURAZZO Marilena
Department of Internal Medicine, University of Turin
-
NIRO Grazia
Department of Gastroenterology, Hospital "Casa Sollievo della Sofferenza"
-
PREMOLI Alberto
Department of Internal Medicine, University of Turin
-
MORELLO Enrico
Department of Internal Medicine, University of Turin
-
BO Simona
Department of Internal Medicine, University of Turin
-
MUSSO Giovanni
Department of Internal Medicine, University of Turin
-
CASSADER Maurizio
Department of Internal Medicine, University of Turin
-
PAGANO Gianfranco
Department of Internal Medicine, University of Turin
-
Bo Simona
Department Of Internal Medicine University Of Turin
-
Niro Grazia
Department Of Gastroenterology Hospital "casa Sollievo Della Sofferenza
-
Morello Enrico
Department Of Internal Medicine University Of Turin
-
Musso Giovanni
Department Of Internal Medicine University Of Turin
-
Premoli Alberto
Department Of Internal Medicine University Of Turin
-
Durazzo Marilena
Department Of Internal Medicine University Of Turin
-
Cassader Maurizio
Department Of Internal Medicine University Of Turin
-
Pagano Gianfranco
Department Of Internal Medicine University Of Turin
関連論文
- Intron 2 [IVS2, T-C +4] HFE gene mutation associated with S65C causes alternative RNA splicing and is responsible for iron overload
- Type 1 autoimmune hepatitis and adipokines : new markers for activity and disease progression?
- Hepatic expression of endocannabinoid receptors and their novel polymorphisms in primary biliary cirrhosis