Decreased Aβ secretion by cells expressing familial Alzheimer's disease-linked mutant presenilin 1
スポンサーリンク
概要
- 論文の詳細を見る
- 2007-03-01
著者
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Ichinose Hiroshi
Department Of Life Science Tokyo Institute Of Technology
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Ichinose Hiroshi
Department Of Life Science Graduate School Of Bioscience And Biotechnology Tokyo Institute Of Techno
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MURAYAMA Miyuki
Laboratory for Azeimer's Disease, Brain Science Institate, RIKEN
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Takashima Akihiko
Laboratory For Alzheimer's Disease Riken Brain Science Institute
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SHIMOJO Masafumi
Laboratory for Alzheimer's Disease, RIKEN Brain Science Institute
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SAHARA Naruhiko
Laboratory for Alzheimer's Disease, RIKEN Brain Science Institute
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Sahara Naruhiko
Laboratory For Alzheimer's Disease Riken Brain Science Institute
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Shimojo Masafumi
Laboratory For Alzheimer's Disease Riken Brain Science Institute
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Murayama Miyuki
Laboratory For Alzheimer's Disease Riken Brain Science Institute
関連論文
- Advanced Research on Dopamine Signaling to Develop Drugs for the Treatment of Mental Disorders: Regulation of Dopaminergic Neural Transmission by Tyrosine Hydroxylase Protein at Nerve Terminals
- Involvement of apoptosis and cholinergic dysfunction in Alzheimer's disease
- Discussions on phosphorylated tau and other biochemical markers
- Molecular Mechanism for Pterin-Mediated Inactivation of Tyrosine Hydroxylase : Formation of Insoluble Aggregates of Tyrosine Hydroxylase
- Familial Alzheimer's disease-associated mutations block translocation of full-length presenilin 1 to the nuclear envelope
- Decreased Aβ secretion by cells expressing familial Alzheimer's disease-linked mutant presenilin 1
- Advanced Research on Dopamine Signaling to Develop Drugs for the Treatment of Mental Disorders : Regulation of Dopaminergic Neural Transmission by Tyrosine Hydroxylase Protein at Nerve Terminals
- Drug Development Targeting the Glycogen Synthase Kinase-3β (GSK-3β)-Mediated Signal Transduction Pathway : Role of GSK-3β in Adult Brain