Metabolism of amyloid β peptide and pathogenesis of Alzheimer's disease Towards presymptomatic diagnosis, prevention and therapy
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概要
- 論文の詳細を見る
- 2006-04-01
著者
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Saido Takaomi
Laboratory For Proteolytic Neuroscience Riken Brain Science Institute
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Iwata Nobuhisa
Laboratory For Proteolytic Neuroscience Riken Brain Science Institute
関連論文
- Distribution of Geriatric Disease-Related Genotypes in the National Institute for Longevity Sciences, Longitudinal Study of Aging (NILS-LSA)
- Autolysis of Calpain Large Subunit Inducing Irreversible Dissociation of Stoichiometric Heterodimer of Calpain
- Thimet Oligopeptidase Cleaves the Full-Length Alzheimer Amyloid Precursor Protein at a β-Secretase Cleavage Site in COS Cells^1
- Involvement of Calpain in Osteoclastic Bone Resorption
- Aβ-degrading endopeptidase, neprilysin, in mouse brain : synaptic and axonal localization inversely correlating with Aβ pathology
- Biochemical Identification of the Neutral Endopeptidase Family Membeer Responsible for the Catabolism of Amyloid β Peptide in the Brain^1
- Metabolism of amyloid β peptide and pathogenesis of Alzheimer's disease Towards presymptomatic diagnosis, prevention and therapy
- Clearance of Extracellular and Cell-Associated Amyloid β Peptide through Viral Expression of Neprilysin in Primary Neurons
- Animal models of tauopathies
- Thimet Oligopeptidase Cleaves the Full-Length Alzheimer Amyloid Precursor Protein at a .BETA.-Secretase Cleavage Site in COS Cells.
- Clearance of Extracellular and Cell-Associated Amyloid .BETA. Peptide through Viral Expression of Neprilysin in Primary Neurons.