RUNX1/EVI1, which blocks myeloid differentiation, inhibits CCAAT-enhancer binding protein α function
スポンサーリンク
概要
- 論文の詳細を見る
- 2007-11-10
著者
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MITANI Kinuko
Department of Hematology, Dokkyo University School of Medicine
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MAKI Kazuhiro
Department of Hematology, Dokkyo Medical University School of Medicine
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Maki Kazuhiro
Department Of Hematology Dokkyo Medical University School Of Medicine
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TOKITA Katsuya
Department of Hematology, Dokkyo Medical University School of Medicine
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Mitani Kinuko
Department Of Hematology Dokkyo Medical University School Of Medicine
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Mitani Kinuko
Department Of Hematology Dokkyo Medical University
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Tokita Katsuya
Department Of Hematology Dokkyo Medical University School Of Medicine
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MAKI Kazuhiro
Department of Hematology and Oncology, Dokkyo Medical University School of Medicine
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MITANI Kinuko
Department of Hematology and Oncology, Dokkyo Medical University School of Medicine
関連論文
- Allogeneic stem cell transplantation versus chemotherapy as post-remission therapy for intermediate or poor risk adult acute myeloid leukemia : results of the JALSG AML97 study
- Enhanced expression of the EVI1 gene in NUP98/HOXA-expressing leukemia cells
- Role of the RUNX1-EVI1 fusion gene in leukemogenesis
- RUNX1/EVI1, which blocks myeloid differentiation, inhibits CCAAT-enhancer binding protein α function
- Phase I/II study of humanized anti-CD33 antibody conjugated with calicheamicin, gemtuzumab ozogamicin, in relapsed or refractory acute myeloid leukemia : final results of Japanese multicenter cooperative study
- A Prospective Study of Cyclosporine A Treatment of Patients with Low-Risk Myelodysplastic Syndrome : Presence of CD55^-CD59^- Blood Cells Predicts Platelet Response
- Leukemia-related transcription factor TEL/ETV6 expands erythroid precursors and stimulates hemoglobin synthesis
- Histone deacetylase inhibitors trichostatin A and valproic acid circumvent apoptosis in human leukemic cells expressing the RUNX1 chimera
- Low-Level Expression of ETV6/TEL in Patients with Myelodysplastic Syndrome
- TEL/ETV6 Binds to Corepressor KAP1 via the HLH Domain
- Runx1/AML1 in Normal and Abnormal Hematopoiesis
- Low Frequency of BCL10 Gene Mutations in B-Cell Non-Hodgkin's Lymphoma
- Presentation of familial Mediterranean fever in a heterozygous MEFV mutation triggered by immunosuppressive therapy for myelodysplastic syndrome
- Chromosomal abnormalities and oncogenes
- Acute myeloid leukemia with t(7;21)(q11.2;q22) expresses a novel, reversed-sequence RUNX1-DTX2 chimera
- Fadd and Skp2 are possible downstream targets of RUNX1-EVI1