CLONING, SEQUENCING, AND FUNCTIONAL CHARACTERIZATION OF PAF-AH IB1 IN NOD MICE : PAF-AH Ib1 MAY NOT BE A CANDIDATE DIABETES SUSCEPTIBILITY GENE IN Idd4.1
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概要
- 論文の詳細を見る
- 2005-10-01
著者
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Wang Zai-zhao
Center For Biotechnology And Genomic Medicine Medical College Of Georgia
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MI Qing-Sheng
Center for Biotechnology and Genomic Medicine, Medical College of Georgia
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ZHOU Li
Center for Biotechnology and Genomic Medicine, Medical College of Georgia
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GRATTAN Masha
Autoimmunity/Diabetes, Robarts Research Institute
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SIVILOTTI Micheal
Autoimmunity/Diabetes, Robarts Research Institute
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SHE Jin-Xion
Center for Biotechnology and Genomic Medicine, Medical College of Georgia
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DELOVITCH Terry
Autoimmunity/Diabetes, Robarts Research Institute
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Mi Qing-sheng
Center For Biotechnology And Genomic Medicine Medical College Of Georgia
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Grattan Masha
Autoimmunity/diabetes Robarts Research Institute
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She Jin-xion
Center For Biotechnology And Genomic Medicine Medical College Of Georgia
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Delovitch Terry
Autoimmunity/diabetes Robarts Research Institute
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Delovitch Terry
Autoimmunity/diabetes Group The John P. Robarts Research Institute
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Sivilotti Micheal
Autoimmunity/diabetes Robarts Research Institute
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Zhou Li
Center For Biotechnology And Genomic Medicine Medical College Of Georgia
関連論文
- CLONING, SEQUENCING, AND FUNCTIONAL CHARACTERIZATION OF PAF-AH IB1 IN NOD MICE : PAF-AH Ib1 MAY NOT BE A CANDIDATE DIABETES SUSCEPTIBILITY GENE IN Idd4.1
- CD28 co-stimulation restores T cell responsiveness in NOD mice by overcoming deficiencies in Rac-1/p38 mitogen-activated protein kinase signaling and IL-2 and IL-4 gene transcription