HEIGHTENED INTERFERONα/β RESPONSE CAUSES MYELOID CELL DYSFUNCTION AND PROMOTES T1D PATHOGENESIS IN NOD MICE

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概要

• 論文の詳細を見る
• 2005-10-01

著者

• CLARE-SALZLER M.

  University of Florida

• PENG Rui

  University of Florida

• PAEK E.

  University of Florida

• SAMUELS M.

  University of Florida

• TENNYSON N.

  University of Florida

• XIA C.

  University of Florida

関連論文

• UP REGULATION OF FOXP3 EXPRESSION IN MOUSE AND HUMAN Treg IS IL-2/STAT5 DEPENDENT ; IMPLICATIONS FOR THE NOD STAT5 MUTATION IN DIABETES PATHOGENESIS
• EICOSANOID IMBALANCE IN THE NOD MOUSE IS RELATED TO A DYSREGULATION ON SOLUBLE EPOXIDE HYDORLASE AND 15-PGHD EXPRESSION
• HEIGHTENED INTERFERONα/β RESPONSE CAUSES MYELOID CELL DYSFUNCTION AND PROMOTES T1D PATHOGENESIS IN NOD MICE
• PEPTIDE-PULSED DENDRITIC CELL THERAPY REDUCES RESPONSES TO β CELL TARGET ANTIGENS AND PROTECTS NOD RECIPIENTS FROM DIABETES

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