In Situ-Produced 7-Chlorokynurenate Has Different Effects on Evoked Responses in Rats with Limbic Epilepsy in Comparison to Naive Controls
スポンサーリンク
概要
- 論文の詳細を見る
- 2005-11-01
著者
-
Zhang De
Department Of Neurology University Of Virginia
-
Bertram Edward
Department Of Immunology University Of Toronto
-
Schwarcz Robert
Maryland Psychiatric Research Center University Of Maryland School Of Medicine
-
Williamson John
Department Of Medicinal Chemistry School Of Pharmacy University Of Mississippi
-
Wu Hu-qui
Maryland Psychiatric Research Center University Of Maryland School Of Medicine
-
Williamson John
Department Of Neurology University Of Virginia
関連論文
- NADPH-diaphorase Activity in the Endolymphatic Sac of the Guinea Pig
- NADPH-diaphorase Activity in the Vestibular End Organs of the Guinea Pig
- Hypothalamic Neuronal Loss and Altered Circadian Rhythm of Temperature in a Rat Model of Mesial Temporal Lobe Epilepsy
- Ibotenate Injections into the Pre- and Parasubiculum Provide Partial Protection against Kainate-Induced Epileptic Damage in Layer III of Rat Entorhinal Cortex
- Ultra-performance liquid chromatography-tandem mass spectrometric method for the determination of Artemisinin in rat serum and its application in pharmacokinetics
- Functional Anatomy of Spontaneous Seizures in a Rat Model of Limbic Epilepsy
- Kynurenate and 7-Chlorokynurenate Formation in Chronically Epileptic Rats
- Distribution and Initiation of Seizure Activity in a Rat Brain with Subcortical Band Heterotopia
- In Situ-Produced 7-Chlorokynurenate Has Different Effects on Evoked Responses in Rats with Limbic Epilepsy in Comparison to Naive Controls
- Volumetric Magnetic Resonance Imaging Evidence of Bilateral Hippocampal Atrophy in Mesial Temporal Lobe Epilepsy
- Overexpression of rab7 enhances the kinetics of antigen processing and presentation with MHC class II molecules in B cells
- The Midline Thalamus : Alterations and a Potential Role in Limbic Epilepsy
- Depression in Intractable Partial Epilepsy Varies by Laterality of Focus and Surgery
- The Pathological Substrate of Limbic Epilepsy : Neuronal Loss in the Medial Dorsal Thalamic Nucleus as the Consistent Change